Tommasino M - Search Results

1

p53 mutations are common in human papillomavirus type 38-positive non-melanoma skin cancers.

Caldeira S, Filotico R, Accardi R, Zehbe I, Franceschi S, Tommasino M. Caldeira S, et al. Among authors: tommasino m. Cancer Lett. 2004 Jun 8;209(1):119-24. doi: 10.1016/j.canlet.2003.12.003. Cancer Lett. 2004. PMID: 15145527

2

Human papillomavirus 16 E6 variants are more prevalent in invasive cervical carcinoma than the prototype.

Zehbe I, Wilander E, Delius H, Tommasino M. Zehbe I, et al. Among authors: tommasino m. Cancer Res. 1998 Feb 15;58(4):829-33. Cancer Res. 1998. PMID: 9485042

3

Risk of cervical cancer and geographical variations of human papillomavirus 16 E6 polymorphisms.

Zehbe I, Voglino G, Delius H, Wilander E, Tommasino M. Zehbe I, et al. Among authors: tommasino m. Lancet. 1998 Oct 31;352(9138):1441-2. doi: 10.1016/S0140-6736(05)61263-9. Lancet. 1998. PMID: 9807995 No abstract available.

4

Overriding of cyclin-dependent kinase inhibitors by high and low risk human papillomavirus types: evidence for an in vivo role in cervical lesions.

Zehbe I, Rätsch A, Alunni-Fabbroni M, Burzlaff A, Bakos E, Dürst M, Wilander E, Tommasino M. Zehbe I, et al. Among authors: tommasino m. Oncogene. 1999 Apr 1;18(13):2201-11. doi: 10.1038/sj.onc.1202549. Oncogene. 1999. PMID: 10327066

5

Codon 72 polymorphism of p53 and its association with cervical cancer.

Zehbe I, Voglino G, Wilander E, Genta F, Tommasino M. Zehbe I, et al. Among authors: tommasino m. Lancet. 1999 Jul 17;354(9174):218-9. doi: 10.1016/S0140-6736(99)01914-5. Lancet. 1999. PMID: 10421306

6

Human papillomavirus E7 proteins stimulate proliferation independently of their ability to associate with retinoblastoma protein.

Caldeira S, de Villiers EM, Tommasino M. Caldeira S, et al. Among authors: tommasino m. Oncogene. 2000 Feb 10;19(6):821-6. doi: 10.1038/sj.onc.1203375. Oncogene. 2000. PMID: 10698500

7

Induction of S phase and apoptosis by the human papillomavirus type 16 E7 protein are separable events in immortalized rodent fibroblasts.

Alunni-Fabbroni M, Littlewood T, Deleu L, Caldeira S, Giarrè M, Dell' Orco M, Tommasino M. Alunni-Fabbroni M, et al. Among authors: tommasino m. Oncogene. 2000 May 4;19(19):2277-85. doi: 10.1038/sj.onc.1203570. Oncogene. 2000. PMID: 10822378

8

Na+/H+ exchanger-dependent intracellular alkalinization is an early event in malignant transformation and plays an essential role in the development of subsequent transformation-associated phenotypes.

Reshkin SJ, Bellizzi A, Caldeira S, Albarani V, Malanchi I, Poignee M, Alunni-Fabbroni M, Casavola V, Tommasino M. Reshkin SJ, et al. Among authors: tommasino m. FASEB J. 2000 Nov;14(14):2185-97. doi: 10.1096/fj.00-0029com. FASEB J. 2000. PMID: 11053239

9

p53 codon 72 polymorphism and various human papillomavirus 16 E6 genotypes are risk factors for cervical cancer development.

Zehbe I, Voglino G, Wilander E, Delius H, Marongiu A, Edler L, Klimek F, Andersson S, Tommasino M. Zehbe I, et al. Among authors: tommasino m. Cancer Res. 2001 Jan 15;61(2):608-11. Cancer Res. 2001. PMID: 11212257

10

Induction of pRb degradation by the human papillomavirus type 16 E7 protein is essential to efficiently overcome p16INK4a-imposed G1 cell cycle Arrest.

Giarrè M, Caldeira S, Malanchi I, Ciccolini F, Leão MJ, Tommasino M. Giarrè M, et al. Among authors: tommasino m. J Virol. 2001 May;75(10):4705-12. doi: 10.1128/JVI.75.10.4705-4712.2001. J Virol. 2001. PMID: 11312342 Free PMC article.

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