Vistica BP - Search Results

1

A unique pattern of up- and down-regulation of chemokine receptor CXCR3 on inflammation-inducing Th1 cells.

Chen J, Vistica BP, Takase H, Ham DI, Fariss RN, Wawrousek EF, Chan CC, DeMartino JA, Farber JM, Gery I. Chen J, et al. Among authors: vistica bp. Eur J Immunol. 2004 Oct;34(10):2885-94. doi: 10.1002/eji.200425318. Eur J Immunol. 2004. PMID: 15368305 Free article.

2

Induction of ocular inflammation by T-helper lymphocytes type 2.

Kim SJ, Zhang M, Vistica BP, Chan CC, Shen DF, Wawrousek EF, Gery I. Kim SJ, et al. Among authors: vistica bp. Invest Ophthalmol Vis Sci. 2002 Mar;43(3):758-65. Invest Ophthalmol Vis Sci. 2002. PMID: 11867595

3

Inflammatory processes triggered by TCR engagement or by local cytokine expression: differences in profiles of gene expression and infiltrating cell populations.

Takase H, Yu CR, Ham DI, Chan CC, Chen J, Vistica BP, Wawrousek EF, Durum SK, Egwuagu CE, Gery I. Takase H, et al. Among authors: vistica bp. J Leukoc Biol. 2006 Sep;80(3):538-45. doi: 10.1189/jlb.1205719. Epub 2006 Jun 22. J Leukoc Biol. 2006. PMID: 16793919

4

Active participation of antigen-nonspecific lymphoid cells in immune-mediated inflammation.

Chen J, Fujimoto C, Vistica BP, He J, Wawrousek EF, Kelsall B, Gery I. Chen J, et al. Among authors: vistica bp. J Immunol. 2006 Sep 1;177(5):3362-8. doi: 10.4049/jimmunol.177.5.3362. J Immunol. 2006. PMID: 16920977

5

Pertussis toxin is superior to TLR ligands in enhancing pathogenic autoimmunity, targeted at a neo-self antigen, by triggering robust expansion of Th1 cells and their cytokine production.

Fujimoto C, Yu CR, Shi G, Vistica BP, Wawrousek EF, Klinman DM, Chan CC, Egwuagu CE, Gery I. Fujimoto C, et al. Among authors: vistica bp. J Immunol. 2006 Nov 15;177(10):6896-903. doi: 10.4049/jimmunol.177.10.6896. J Immunol. 2006. PMID: 17082604

6

Both Th1 and Th17 are immunopathogenic but differ in other key biological activities.

Cox CA, Shi G, Yin H, Vistica BP, Wawrousek EF, Chan CC, Gery I. Cox CA, et al. Among authors: vistica bp. J Immunol. 2008 Jun 1;180(11):7414-22. doi: 10.4049/jimmunol.180.11.7414. J Immunol. 2008. PMID: 18490741 Free PMC article.

7

Phenotype switching by inflammation-inducing polarized Th17 cells, but not by Th1 cells.

Shi G, Cox CA, Vistica BP, Tan C, Wawrousek EF, Gery I. Shi G, et al. Among authors: vistica bp. J Immunol. 2008 Nov 15;181(10):7205-13. doi: 10.4049/jimmunol.181.10.7205. J Immunol. 2008. PMID: 18981142 Free PMC article.

8

Unlike Th1, Th17 cells mediate sustained autoimmune inflammation and are highly resistant to restimulation-induced cell death.

Shi G, Ramaswamy M, Vistica BP, Cox CA, Tan C, Wawrousek EF, Siegel RM, Gery I. Shi G, et al. Among authors: vistica bp. J Immunol. 2009 Dec 1;183(11):7547-56. doi: 10.4049/jimmunol.0900519. Epub 2009 Nov 4. J Immunol. 2009. PMID: 19890052 Free PMC article.

9

Antigen-specific Th9 cells exhibit uniqueness in their kinetics of cytokine production and short retention at the inflammatory site.

Tan C, Aziz MK, Lovaas JD, Vistica BP, Shi G, Wawrousek EF, Gery I. Tan C, et al. Among authors: vistica bp. J Immunol. 2010 Dec 1;185(11):6795-801. doi: 10.4049/jimmunol.1001676. Epub 2010 Oct 22. J Immunol. 2010. PMID: 20971929 Free PMC article.

10

Inflammation-inducing Th1 and Th17 cells differ in their expression patterns of apoptosis-related molecules.

Tan C, Ramaswamy M, Shi G, Vistica BP, Siegel RM, Gery I. Tan C, et al. Among authors: vistica bp. Cell Immunol. 2011;271(2):210-3. doi: 10.1016/j.cellimm.2011.08.006. Epub 2011 Aug 17. Cell Immunol. 2011. PMID: 21903206 Free PMC article.

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