Lundkvist J - Search Results

1

Detection of the founder effect in Finnish CADASIL families.

Mykkänen K, Savontaus ML, Juvonen V, Sistonen P, Tuisku S, Tuominen S, Penttinen M, Lundkvist J, Viitanen M, Kalimo H, Pöyhönen M. Mykkänen K, et al. Among authors: lundkvist j. Eur J Hum Genet. 2004 Oct;12(10):813-9. doi: 10.1038/sj.ejhg.5201221. Eur J Hum Genet. 2004. PMID: 15378071

2

Mice carrying a R142C Notch 3 knock-in mutation do not develop a CADASIL-like phenotype.

Lundkvist J, Zhu S, Hansson EM, Schweinhardt P, Miao Q, Beatus P, Dannaeus K, Karlström H, Johansson CB, Viitanen M, Rozell B, Spenger C, Mohammed A, Kalimo H, Lendahl U. Lundkvist J, et al. Genesis. 2005 Jan;41(1):13-22. doi: 10.1002/gene.20091. Genesis. 2005. PMID: 15645445

3

A CADASIL-mutated Notch 3 receptor exhibits impaired intracellular trafficking and maturation but normal ligand-induced signaling.

Karlström H, Beatus P, Dannaeus K, Chapman G, Lendahl U, Lundkvist J. Karlström H, et al. Among authors: lundkvist j. Proc Natl Acad Sci U S A. 2002 Dec 24;99(26):17119-24. doi: 10.1073/pnas.252624099. Epub 2002 Dec 13. Proc Natl Acad Sci U S A. 2002. PMID: 12482954 Free PMC article.

4

The notch 3 intracellular domain represses notch 1-mediated activation through Hairy/Enhancer of split (HES) promoters.

Beatus P, Lundkvist J, Oberg C, Lendahl U. Beatus P, et al. Among authors: lundkvist j. Development. 1999 Sep;126(17):3925-35. doi: 10.1242/dev.126.17.3925. Development. 1999. PMID: 10433920

5

The origin of the ankyrin repeat region in Notch intracellular domains is critical for regulation of HES promoter activity.

Beatus P, Lundkvist J, Oberg C, Pedersen K, Lendahl U. Beatus P, et al. Among authors: lundkvist j. Mech Dev. 2001 Jun;104(1-2):3-20. doi: 10.1016/s0925-4773(01)00373-2. Mech Dev. 2001. PMID: 11404076 Free article.

6

Notch and the birth of glial cells.

Lundkvist J, Lendahl U. Lundkvist J, et al. Trends Neurosci. 2001 Sep;24(9):492-4. doi: 10.1016/s0166-2236(00)01888-9. Trends Neurosci. 2001. PMID: 11506867 Review.

7

A sensitive and quantitative assay for measuring cleavage of presenilin substrates.

Karlström H, Bergman A, Lendahl U, Näslund J, Lundkvist J. Karlström H, et al. Among authors: lundkvist j. J Biol Chem. 2002 Mar 1;277(9):6763-6. doi: 10.1074/jbc.C100649200. Epub 2001 Dec 13. J Biol Chem. 2002. PMID: 11744687 Free article.

8

Notch receptor cleavage depends on but is not directly executed by presenilins.

Taniguchi Y, Karlström H, Lundkvist J, Mizutani T, Otaka A, Vestling M, Bernstein A, Donoviel D, Lendahl U, Honjo T. Taniguchi Y, et al. Among authors: lundkvist j. Proc Natl Acad Sci U S A. 2002 Mar 19;99(6):4014-9. doi: 10.1073/pnas.052017699. Epub 2002 Mar 12. Proc Natl Acad Sci U S A. 2002. PMID: 11891288 Free PMC article.

9

APP intracellular domain formation and unaltered signaling in the presence of familial Alzheimer's disease mutations.

Bergman A, Religa D, Karlström H, Laudon H, Winblad B, Lannfelt L, Lundkvist J, Näslund J. Bergman A, et al. Among authors: lundkvist j. Exp Cell Res. 2003 Jul 1;287(1):1-9. doi: 10.1016/s0014-4827(03)00117-4. Exp Cell Res. 2003. PMID: 12799176

10

Pen-2 is sequestered in the endoplasmic reticulum and subjected to ubiquitylation and proteasome-mediated degradation in the absence of presenilin.

Bergman A, Hansson EM, Pursglove SE, Farmery MR, Lannfelt L, Lendahl U, Lundkvist J, Näslund J. Bergman A, et al. Among authors: lundkvist j. J Biol Chem. 2004 Apr 16;279(16):16744-53. doi: 10.1074/jbc.M313999200. Epub 2004 Jan 14. J Biol Chem. 2004. PMID: 14724271 Free article.

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