Akagawa KS - Search Results

1

Abnormally differentiated subsets of intestinal macrophage play a key role in Th1-dominant chronic colitis through excess production of IL-12 and IL-23 in response to bacteria.

Kamada N, Hisamatsu T, Okamoto S, Sato T, Matsuoka K, Arai K, Nakai T, Hasegawa A, Inoue N, Watanabe N, Akagawa KS, Hibi T. Kamada N, et al. Among authors: akagawa ks. J Immunol. 2005 Nov 15;175(10):6900-8. doi: 10.4049/jimmunol.175.10.6900. J Immunol. 2005. PMID: 16272349

2

Unique CD14 intestinal macrophages contribute to the pathogenesis of Crohn disease via IL-23/IFN-gamma axis.

Kamada N, Hisamatsu T, Okamoto S, Chinen H, Kobayashi T, Sato T, Sakuraba A, Kitazume MT, Sugita A, Koganei K, Akagawa KS, Hibi T. Kamada N, et al. Among authors: akagawa ks. J Clin Invest. 2008 Jun;118(6):2269-80. doi: 10.1172/JCI34610. J Clin Invest. 2008. PMID: 18497880 Free PMC article.

3

IL-10 inhibits granulocyte-macrophage colony-stimulating factor-dependent human monocyte survival at the early stage of the culture and inhibits the generation of macrophages.

Hashimoto SI, Komuro I, Yamada M, Akagawa KS. Hashimoto SI, et al. Among authors: akagawa ks. J Immunol. 2001 Oct 1;167(7):3619-25. doi: 10.4049/jimmunol.167.7.3619. J Immunol. 2001. PMID: 11564774

4

Suppression of TNF-alpha mRNA expression in LPS-primed macrophages occurs at the level of nuclear factor-kappa B activation, but not at the level of protein kinase C or CD14 expression.

Takasuka N, Matsuura K, Yamamoto S, Akagawa KS. Takasuka N, et al. Among authors: akagawa ks. J Immunol. 1995 May 1;154(9):4803-12. J Immunol. 1995. PMID: 7536782

5

Granulocyte-macrophage colony-stimulating factor promotes the proliferation of human alveolar macrophages in vitro.

Nakata K, Akagawa KS, Fukayama M, Hayashi Y, Kadokura M, Tokunaga T. Nakata K, et al. Among authors: akagawa ks. J Immunol. 1991 Aug 15;147(4):1266-72. J Immunol. 1991. PMID: 1869822

6

Effects of granulocyte-macrophage colony-stimulating factor and colony-stimulating factor-1 on the proliferation and differentiation of murine alveolar macrophages.

Akagawa KS, Kamoshita K, Tokunaga T. Akagawa KS, et al. J Immunol. 1988 Nov 15;141(10):3383-90. J Immunol. 1988. PMID: 3053898

7

Preexposure of macrophages to low doses of lipopolysaccharide inhibits the expression of tumor necrosis factor-alpha mRNA but not of IL-1 beta mRNA.

Takasuka N, Tokunaga T, Akagawa KS. Takasuka N, et al. Among authors: akagawa ks. J Immunol. 1991 Jun 1;146(11):3824-30. J Immunol. 1991. PMID: 1903414

8

Lipopolysaccharide-induced biphasic inositol 1,4,5-trisphosphate response and tyrosine phosphorylation of 140-kilodalton protein in mouse peritoneal macrophages.

Shinji H, Akagawa KS, Tsuji M, Maeda M, Yamada R, Matsuura K, Yamamoto S, Yoshida T. Shinji H, et al. Among authors: akagawa ks. J Immunol. 1997 Feb 1;158(3):1370-6. J Immunol. 1997. PMID: 9013981

9

LPS induces selective translocation of protein kinase C-beta in LPS-responsive mouse macrophages, but not in LPS-nonresponsive mouse macrophages.

Shinji H, Akagawa KS, Yoshida T. Shinji H, et al. Among authors: akagawa ks. J Immunol. 1994 Dec 15;153(12):5760-71. J Immunol. 1994. PMID: 7989773

10

Interleukin-10 contributes development of macrophage suppressor activities by macrophage colony-stimulating factor, but not by granulocyte-macrophage colony-stimulating factor.

Mochida-Nishimura K, Akagawa KS, Rich EA. Mochida-Nishimura K, et al. Among authors: akagawa ks. Cell Immunol. 2001 Nov 25;214(1):81-8. doi: 10.1006/cimm.2001.1801. Cell Immunol. 2001. PMID: 11902832

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