Chesnokova LS - Search Results

1

Antibodies to gp350/220 enhance the ability of Epstein-Barr virus to infect epithelial cells.

Turk SM, Jiang R, Chesnokova LS, Hutt-Fletcher LM. Turk SM, et al. Among authors: chesnokova ls. J Virol. 2006 Oct;80(19):9628-33. doi: 10.1128/JVI.00622-06. J Virol. 2006. PMID: 16973566 Free PMC article.

2

Fusion of epithelial cells by Epstein-Barr virus proteins is triggered by binding of viral glycoproteins gHgL to integrins alphavbeta6 or alphavbeta8.

Chesnokova LS, Nishimura SL, Hutt-Fletcher LM. Chesnokova LS, et al. Proc Natl Acad Sci U S A. 2009 Dec 1;106(48):20464-9. doi: 10.1073/pnas.0907508106. Epub 2009 Nov 17. Proc Natl Acad Sci U S A. 2009. PMID: 19920174 Free PMC article.

3

Integrins as triggers of Epstein-Barr virus fusion and epithelial cell infection.

Hutt-Fletcher LM, Chesnokova LS. Hutt-Fletcher LM, et al. Among authors: chesnokova ls. Virulence. 2010 Sep-Oct;1(5):395-8. doi: 10.4161/viru.1.5.12546. Virulence. 2010. PMID: 21178476 Free PMC article. Review.

4

Fusion of Epstein-Barr virus with epithelial cells can be triggered by αvβ5 in addition to αvβ6 and αvβ8, and integrin binding triggers a conformational change in glycoproteins gHgL.

Chesnokova LS, Hutt-Fletcher LM. Chesnokova LS, et al. J Virol. 2011 Dec;85(24):13214-23. doi: 10.1128/JVI.05580-11. Epub 2011 Sep 28. J Virol. 2011. PMID: 21957301 Free PMC article.

5

αvβ3-integrin is a major sensor and activator of innate immunity to herpes simplex virus-1.

Gianni T, Leoni V, Chesnokova LS, Hutt-Fletcher LM, Campadelli-Fiume G. Gianni T, et al. Among authors: chesnokova ls. Proc Natl Acad Sci U S A. 2012 Nov 27;109(48):19792-7. doi: 10.1073/pnas.1212597109. Epub 2012 Nov 12. Proc Natl Acad Sci U S A. 2012. PMID: 23150579 Free PMC article.

6

αvβ6- and αvβ8-integrins serve as interchangeable receptors for HSV gH/gL to promote endocytosis and activation of membrane fusion.

Gianni T, Salvioli S, Chesnokova LS, Hutt-Fletcher LM, Campadelli-Fiume G. Gianni T, et al. Among authors: chesnokova ls. PLoS Pathog. 2013;9(12):e1003806. doi: 10.1371/journal.ppat.1003806. Epub 2013 Dec 19. PLoS Pathog. 2013. PMID: 24367260 Free PMC article.

7

Epstein-Barr virus glycoprotein gB and gHgL can mediate fusion and entry in trans, and heat can act as a partial surrogate for gHgL and trigger a conformational change in gB.

Chesnokova LS, Ahuja MK, Hutt-Fletcher LM. Chesnokova LS, et al. J Virol. 2014 Nov;88(21):12193-201. doi: 10.1128/JVI.01597-14. Epub 2014 Aug 20. J Virol. 2014. PMID: 25142593 Free PMC article.

8

Epstein-Barr virus infection mechanisms.

Chesnokova LS, Hutt-Fletcher LM. Chesnokova LS, et al. Chin J Cancer. 2014 Nov;33(11):545-8. doi: 10.5732/cjc.014.10168. Epub 2014 Oct 17. Chin J Cancer. 2014. PMID: 25322867 Free PMC article. Review.

9

Viral Entry.

Chesnokova LS, Jiang R, Hutt-Fletcher LM. Chesnokova LS, et al. Curr Top Microbiol Immunol. 2015;391:221-35. doi: 10.1007/978-3-319-22834-1_7. Curr Top Microbiol Immunol. 2015. PMID: 26428376 Review.

10

The BDLF3 gene product of Epstein-Barr virus, gp150, mediates non-productive binding to heparan sulfate on epithelial cells and only the binding domain of CD21 is required for infection.

Chesnokova LS, Valencia SM, Hutt-Fletcher LM. Chesnokova LS, et al. Virology. 2016 Jul;494:23-8. doi: 10.1016/j.virol.2016.04.002. Epub 2016 Apr 8. Virology. 2016. PMID: 27061054 Free PMC article.

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