Vlaminckx E - Search Results

1

In-vitro experimental models for the risk assessment of antibiotic-induced QT prolongation.

Lu HR, Vlaminckx E, Van de Water A, Rohrbacher J, Hermans A, Gallacher DJ. Lu HR, et al. Among authors: vlaminckx e. Eur J Pharmacol. 2007 Dec 22;577(1-3):222-32. doi: 10.1016/j.ejphar.2007.07.070. Eur J Pharmacol. 2007. PMID: 18074444

2

Drug-induced long QT in isolated rabbit Purkinje fibers: importance of action potential duration, triangulation and early afterdepolarizations.

Lu HR, Vlaminckx E, Van Ammel K, De Clerck F. Lu HR, et al. Among authors: vlaminckx e. Eur J Pharmacol. 2002 Oct 4;452(2):183-92. doi: 10.1016/s0014-2999(02)02246-x. Eur J Pharmacol. 2002. PMID: 12354568

3

Choice of cardiac tissue plays an important role in the evaluation of drug-induced prolongation of the QT interval in vitro in rabbit.

Lu HR, Vlaminckx E, Teisman A, Gallacher DJ. Lu HR, et al. Among authors: vlaminckx e. J Pharmacol Toxicol Methods. 2005 Jul-Aug;52(1):90-105. doi: 10.1016/j.vascn.2005.04.007. J Pharmacol Toxicol Methods. 2005. PMID: 15978848

4

Calmodulin antagonist W-7 prevents sparfloxacin-induced early afterdepolarizations (EADs) in isolated rabbit purkinje fibers: importance of beat-to-beat instability of the repolarization.

Lu HR, Vlaminckx E, Van de Water A, Gallacher DJ. Lu HR, et al. Among authors: vlaminckx e. J Cardiovasc Electrophysiol. 2006 Apr;17(4):415-22. doi: 10.1111/j.1540-8167.2006.00420.x. J Cardiovasc Electrophysiol. 2006. PMID: 16643366

5

In-vitro experimental models for the risk assessment of antibiotic-induced QT prolongation.

Lu HR, Vlaminckx E, Van de Water A, Rohrbacher J, Hermans A, Gallacher DJ. Lu HR, et al. Among authors: vlaminckx e. Eur J Pharmacol. 2006 Dec 28;553(1-3):229-39. doi: 10.1016/j.ejphar.2006.09.035. Epub 2006 Sep 23. Eur J Pharmacol. 2006. Corrected and republished in: Eur J Pharmacol. 2007 Dec 22;577(1-3):222-32. doi: 10.1016/j.ejphar.2007.07.070 PMID: 17054943 Corrected and republished.

6

Choice of cardiac tissue in vitro plays an important role in assessing the risk of drug-induced cardiac arrhythmias in human: beyond QT prolongation.

Lu HR, Vlaminckx E, Gallacher DJ. Lu HR, et al. Among authors: vlaminckx e. J Pharmacol Toxicol Methods. 2008 Jan-Feb;57(1):1-8. doi: 10.1016/j.vascn.2007.06.005. Epub 2007 Jul 17. J Pharmacol Toxicol Methods. 2008. PMID: 17964190

7

Predicting drug-induced slowing of conduction and pro-arrhythmia: identifying the 'bad' sodium current blockers.

Lu HR, Rohrbacher J, Vlaminckx E, Van Ammel K, Yan GX, Gallacher DJ. Lu HR, et al. Among authors: vlaminckx e. Br J Pharmacol. 2010 May;160(1):60-76. doi: 10.1111/j.1476-5381.2010.00646.x. Epub 2010 Mar 22. Br J Pharmacol. 2010. PMID: 20331615 Free PMC article.

8

Direct effects of arsenic trioxide on action potentials in isolated cardiac tissues: importance of the choice of species, type of cardiac tissue and perfusion time.

Lu HR, Vlaminckx E, Cools F, Gallacher DJ. Lu HR, et al. Among authors: vlaminckx e. J Pharmacol Toxicol Methods. 2012 Sep;66(2):135-44. doi: 10.1016/j.vascn.2012.03.002. Epub 2012 Mar 15. J Pharmacol Toxicol Methods. 2012. PMID: 22445855

9

Both beta-adrenergic receptor stimulation and cardiac tissue type have important roles in elucidating the functional effects of I(Ks) channel blockers in vitro.

Lu HR, Vlaminckx E, Van De Water A, Gallacher DJ. Lu HR, et al. Among authors: vlaminckx e. J Pharmacol Toxicol Methods. 2005 Mar-Apr;51(2):81-90. doi: 10.1016/j.vascn.2004.10.004. J Pharmacol Toxicol Methods. 2005. PMID: 15767201

10

Predicting drug-induced changes in QT interval and arrhythmias: QT-shortening drugs point to gaps in the ICHS7B Guidelines.

Lu HR, Vlaminckx E, Hermans AN, Rohrbacher J, Van Ammel K, Towart R, Pugsley M, Gallacher DJ. Lu HR, et al. Among authors: vlaminckx e. Br J Pharmacol. 2008 Aug;154(7):1427-38. doi: 10.1038/bjp.2008.191. Epub 2008 May 19. Br J Pharmacol. 2008. PMID: 18493243 Free PMC article.

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