Willems PH - Search Results

1

Mitigation of NADH: ubiquinone oxidoreductase deficiency by chronic Trolox treatment.

Koopman WJ, Verkaart S, van Emst-de Vries SE, Grefte S, Smeitink JA, Nijtmans LG, Willems PH. Koopman WJ, et al. Among authors: willems ph. Biochim Biophys Acta. 2008 Jul-Aug;1777(7-8):853-9. doi: 10.1016/j.bbabio.2008.03.028. Epub 2008 Apr 8. Biochim Biophys Acta. 2008. PMID: 18435906 Free article.

2

The diacylglycerol kinase inhibitor, R59022, potentiates cholecystokinin-induced enzyme secretion from rabbit pancreatic acini.

Ederveen AG, van Emst-de Vries SE, De Pont JJ, Willems PH. Ederveen AG, et al. Among authors: willems ph. Eur J Biochem. 1990 Mar 10;188(2):333-8. doi: 10.1111/j.1432-1033.1990.tb15408.x. Eur J Biochem. 1990. PMID: 1690650 Free article.

3

Ederveen AG, Van Emst-De Vries SE, de Pont JJ, Willems PH. Ederveen AG, et al. Among authors: willems ph. Eur J Biochem. 1990 Oct 5;193(1):291-5. doi: 10.1111/j.1432-1033.1990.tb19335.x. Eur J Biochem. 1990. PMID: 1699756 Free article.

4

Effects of phorbol ester and cholecystokinin on the intracellular distribution of protein kinase C in rabbit pancreatic acini.

Ederveen AG, van Emst-de Vries SE, de Pont JJ, Willems PH. Ederveen AG, et al. Among authors: willems ph. Eur J Biochem. 1991 Feb 14;195(3):679-83. doi: 10.1111/j.1432-1033.1991.tb15753.x. Eur J Biochem. 1991. PMID: 1705508 Free article.

5

Cholecystokinin-stimulated enzyme secretion from dispersed rabbit pancreatic acinar cells: phosphorylation-dependent changes in potency and efficacy.

Willems PH, Van Emst-de Vries SE, De Pont JJ. Willems PH, et al. Pflugers Arch. 1995 Sep;430(5):626-35. doi: 10.1007/BF00386156. Pflugers Arch. 1995. PMID: 7478913 Free article.

6

Dose-dependent recruitment of pancreatic acinar cells during receptor-mediated calcium mobilization.

Willems PH, Van Emst-De Vries SE, Van Os CH, De Pont JJ. Willems PH, et al. Cell Calcium. 1993 Feb;14(2):145-59. doi: 10.1016/0143-4160(93)90084-j. Cell Calcium. 1993. PMID: 7681361

7

Receptor-evoked Ca2+ mobilization in pancreatic acinar cells: evidence for a regulatory role of protein kinase C by a mechanism involving the transition of high-affinity receptors to a low-affinity state.

Willems PH, Van Hoof HJ, Van Mackelenbergh MG, Hoenderop JG, Van Emst-De Vries SE, De Pont JJ. Willems PH, et al. Pflugers Arch. 1993 Jul;424(2):171-82. doi: 10.1007/BF00374609. Pflugers Arch. 1993. PMID: 7692387

8

CCK8-evoked Ca2+ mobilization in pancreatic acinar cells. Evidence for a regulatory role of protein kinase C by phosphorylation-dependent inhibition of signaling through the high-affinity CCK receptor.

Willems PH, van Hoof HJ, van Mackelenbergh MG, Hoenderop JG, van Emst-de Vries SE, De Pont JJ. Willems PH, et al. Ann N Y Acad Sci. 1994 Mar 23;713:343-5. doi: 10.1111/j.1749-6632.1994.tb44087.x. Ann N Y Acad Sci. 1994. PMID: 8185182 No abstract available.

9

Recovery from TPA inhibition of receptor-mediated Ca2+ mobilization is paralleled by down-regulation of protein kinase C-alpha in CHO cells expressing the CCK-A receptor.

Smeets RL, Garner KM, Hendriks M, van Emst-de Vries SE, Peacock MD, Hendriks W, de Pont JJ, Willems PH. Smeets RL, et al. Among authors: willems ph. Cell Calcium. 1996 Jul;20(1):1-9. doi: 10.1016/s0143-4160(96)90045-7. Cell Calcium. 1996. PMID: 8864566

10

Mutational analysis of the putative devazepide binding site of the CCK(A) receptor.

Smeets RL, IJzerman AP, Hermsen HP, Ophorst OJ, Van Emst-de Vries SE, De Pont JJ, Willems PH. Smeets RL, et al. Among authors: willems ph. Eur J Pharmacol. 1997 Apr 23;325(1):93-9. doi: 10.1016/s0014-2999(97)00106-4. Eur J Pharmacol. 1997. PMID: 9151944 Free article.

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