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Bacterial colitis increases susceptibility to oral prion disease.
Sigurdson CJ, Heikenwalder M, Manco G, Barthel M, Schwarz P, Stecher B, Krautler NJ, Hardt WD, Seifert B, MacPherson AJ, Corthesy I, Aguzzi A. Sigurdson CJ, et al. Among authors: heikenwalder m. J Infect Dis. 2009 Jan 15;199(2):243-52. doi: 10.1086/595791. J Infect Dis. 2009. PMID: 19072552 Free PMC article.
Prion diseases: Cannibals and garbage piles.
Aguzzi A, Heikenwalder M. Aguzzi A, et al. Among authors: heikenwalder m. Nature. 2003 May 8;423(6936):127-9. doi: 10.1038/423127a. Nature. 2003. PMID: 12736665 No abstract available.
The Salmonella pathogenicity island (SPI)-2 and SPI-1 type III secretion systems allow Salmonella serovar typhimurium to trigger colitis via MyD88-dependent and MyD88-independent mechanisms.
Hapfelmeier S, Stecher B, Barthel M, Kremer M, Müller AJ, Heikenwalder M, Stallmach T, Hensel M, Pfeffer K, Akira S, Hardt WD. Hapfelmeier S, et al. Among authors: heikenwalder m. J Immunol. 2005 Feb 1;174(3):1675-85. doi: 10.4049/jimmunol.174.3.1675. J Immunol. 2005. PMID: 15661931
346 results