Johannessen AC - Search Results

1

Camptothecin and khat (Catha edulis Forsk.) induced distinct cell death phenotypes involving modulation of c-FLIPL, Mcl-1, procaspase-8 and mitochondrial function in acute myeloid leukemia cell lines.

Bredholt T, Dimba EA, Hagland HR, Wergeland L, Skavland J, Fossan KO, Tronstad KJ, Johannessen AC, Vintermyr OK, Gjertsen BT. Bredholt T, et al. Among authors: johannessen ac. Mol Cancer. 2009 Nov 13;8:101. doi: 10.1186/1476-4598-8-101. Mol Cancer. 2009. PMID: 19912650 Free PMC article.

2

Early loss of mitochondrial inner transmembrane potential in khat-induced cell death of primary normal human oral cells.

Lukandu OM, Bredholt T, Neppelberg E, Gjertsen BT, Johannessen AC, Vintermyr OK, Costea DE. Lukandu OM, et al. Among authors: johannessen ac. Toxicology. 2009 Sep 19;263(2-3):108-16. doi: 10.1016/j.tox.2009.06.024. Epub 2009 Jul 8. Toxicology. 2009. PMID: 19591894

3

Catha edulis (Khat) induces cell death by apoptosis in leukemia cell lines.

Dimba E, Gjertsen BT, Francis GW, Johannessen AC, Vintermyr OK. Dimba E, et al. Among authors: johannessen ac. Ann N Y Acad Sci. 2003 Dec;1010:384-8. doi: 10.1196/annals.1299.070. Ann N Y Acad Sci. 2003. PMID: 15033757

4

Khat (Catha edulis)-induced apoptosis is inhibited by antagonists of caspase-1 and -8 in human leukaemia cells.

Dimba EA, Gjertsen BT, Bredholt T, Fossan KO, Costea DE, Francis GW, Johannessen AC, Vintermyr OK. Dimba EA, et al. Among authors: johannessen ac. Br J Cancer. 2004 Nov 1;91(9):1726-34. doi: 10.1038/sj.bjc.6602197. Br J Cancer. 2004. PMID: 15477863 Free PMC article.

5

Khat induces G1-phase arrest and increased expression of stress-sensitive p53 and p16 proteins in normal human oral keratinocytes and fibroblasts.

Lukandu OM, Costea DE, Dimba EA, Neppelberg E, Bredholt T, Gjertsen BT, Vintermyr OK, Johannessen AC. Lukandu OM, et al. Among authors: johannessen ac. Eur J Oral Sci. 2008 Feb;116(1):23-30. doi: 10.1111/j.1600-0722.2007.00508.x. Eur J Oral Sci. 2008. PMID: 18186728

6

Khat (Catha edulis) induces reactive oxygen species and apoptosis in normal human oral keratinocytes and fibroblasts.

Lukandu OM, Costea DE, Neppelberg E, Johannessen AC, Vintermyr OK. Lukandu OM, et al. Among authors: johannessen ac. Toxicol Sci. 2008 Jun;103(2):311-24. doi: 10.1093/toxsci/kfn044. Epub 2008 Feb 29. Toxicol Sci. 2008. PMID: 18310640

7

Khat alters the phenotype of in vitro-reconstructed human oral mucosa.

Lukandu OM, Neppelberg E, Vintermyr OK, Johannessen AC, Costea DE. Lukandu OM, et al. Among authors: johannessen ac. J Dent Res. 2010 Mar;89(3):270-5. doi: 10.1177/0022034509354980. Epub 2009 Dec 31. J Dent Res. 2010. PMID: 20044580

8

Loss of BCL-2 in the progression of oral cancer is not attributable to mutations.

Loro LL, Johannessen AC, Vintermyr OK. Loro LL, et al. Among authors: johannessen ac. J Clin Pathol. 2005 Nov;58(11):1157-62. doi: 10.1136/jcp.2004.021709. J Clin Pathol. 2005. PMID: 16254104 Free PMC article.

9

Oral squamous cell carcinoma is associated with decreased bcl-2/bax expression ratio and increased apoptosis.

Loro LL, Vintermyr OK, Liavaag PG, Jonsson R, Johannessen AC. Loro LL, et al. Among authors: johannessen ac. Hum Pathol. 1999 Sep;30(9):1097-105. doi: 10.1016/s0046-8177(99)90229-0. Hum Pathol. 1999. PMID: 10492046

10

Apoptosis and expression of Bax and Bcl-2 in snuff- and non-snuff associated oral squamous cell carcinomas.

Loro LL, Vintermyr OK, Ibrahim SO, Idris AM, Johannessen AC. Loro LL, et al. Among authors: johannessen ac. Anticancer Res. 2000 Sep-Oct;20(5A):2855-60. Anticancer Res. 2000. PMID: 11062693

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