Vaya J - Search Results

1

Paraoxonase 1 attenuates human plaque atherogenicity: relevance to the enzyme lactonase activity.

Tavori H, Vaya J, Aviram M. Tavori H, et al. Among authors: vaya j. Adv Exp Med Biol. 2010;660:99-111. doi: 10.1007/978-1-60761-350-3_10. Adv Exp Med Biol. 2010. PMID: 20221874 Review.

2

Characterization of the PON1 active site using modeling simulation, in relation to PON1 lactonase activity.

Tavori H, Khatib S, Aviram M, Vaya J. Tavori H, et al. Among authors: vaya j. Bioorg Med Chem. 2008 Aug 1;16(15):7504-9. doi: 10.1016/j.bmc.2008.06.008. Epub 2008 Jun 12. Bioorg Med Chem. 2008. PMID: 18572410

3

Human carotid atherosclerotic plaque increases oxidative state of macrophages and low-density lipoproteins, whereas paraoxonase 1 (PON1) decreases such atherogenic effects.

Tavori H, Aviram M, Khatib S, Musa R, Nitecki S, Hoffman A, Vaya J. Tavori H, et al. Among authors: vaya j. Free Radic Biol Med. 2009 Mar 1;46(5):607-15. doi: 10.1016/j.freeradbiomed.2008.11.019. Epub 2008 Dec 6. Free Radic Biol Med. 2009. PMID: 19103284

4

Human carotid lesion linoleic acid hydroperoxide inhibits paraoxonase 1 (PON1) activity via reaction with PON1 free sulfhydryl cysteine 284.

Tavori H, Aviram M, Khatib S, Musa R, Mannheim D, Karmeli R, Vaya J. Tavori H, et al. Among authors: vaya j. Free Radic Biol Med. 2011 Jan 1;50(1):148-56. doi: 10.1016/j.freeradbiomed.2010.10.708. Epub 2010 Oct 29. Free Radic Biol Med. 2011. PMID: 21044882

5

Paraoxonase 1 protects macrophages from atherogenicity of a specific triglyceride isolated from human carotid lesion.

Tavori H, Aviram M, Khatib S, Musa R, Mannheim D, Karmeli R, Vaya J. Tavori H, et al. Among authors: vaya j. Free Radic Biol Med. 2011 Jul 1;51(1):234-42. doi: 10.1016/j.freeradbiomed.2011.03.041. Epub 2011 Apr 13. Free Radic Biol Med. 2011. PMID: 21530644

6

Paraoxonase 1 activities, regulation, and interactions with atherosclerotic lesion.

Aviram M, Vaya J. Aviram M, et al. Among authors: vaya j. Curr Opin Lipidol. 2013 Aug;24(4):339-44. doi: 10.1097/MOL.0b013e32835ffcfd. Curr Opin Lipidol. 2013. PMID: 23508039 Review.

7

Human carotid plaque phosphatidylcholine specifically interacts with paraoxonase 1, increases its activity, and enhances its uptake by macrophage at the expense of its binding to HDL.

Cohen E, Aviram M, Khatib S, Artoul F, Rabin A, Mannheim D, Karmeli R, Salamon T, Vaya J. Cohen E, et al. Among authors: vaya j. Free Radic Biol Med. 2014 Nov;76:14-24. doi: 10.1016/j.freeradbiomed.2014.07.036. Epub 2014 Aug 1. Free Radic Biol Med. 2014. PMID: 25091896

8

Human carotid atherosclerotic plaque protein(s) change HDL protein(s) composition and impair HDL anti-oxidant activity.

Cohen E, Aviram M, Khatib S, Volkova N, Vaya J. Cohen E, et al. Among authors: vaya j. Biofactors. 2016 Jan-Feb;42(1):115-28. doi: 10.1002/biof.1254. Epub 2015 Dec 11. Biofactors. 2016. PMID: 26662883

9

Paraoxonase 1 (PON1) enhances HDL-mediated macrophage cholesterol efflux via the ABCA1 transporter in association with increased HDL binding to the cells: a possible role for lysophosphatidylcholine.

Rosenblat M, Vaya J, Shih D, Aviram M. Rosenblat M, et al. Among authors: vaya j. Atherosclerosis. 2005 Mar;179(1):69-77. doi: 10.1016/j.atherosclerosis.2004.10.028. Epub 2004 Dec 29. Atherosclerosis. 2005. PMID: 15721011

10

The catalytic histidine dyad of high density lipoprotein-associated serum paraoxonase-1 (PON1) is essential for PON1-mediated inhibition of low density lipoprotein oxidation and stimulation of macrophage cholesterol efflux.

Rosenblat M, Gaidukov L, Khersonsky O, Vaya J, Oren R, Tawfik DS, Aviram M. Rosenblat M, et al. Among authors: vaya j. J Biol Chem. 2006 Mar 17;281(11):7657-65. doi: 10.1074/jbc.M512595200. Epub 2006 Jan 10. J Biol Chem. 2006. PMID: 16407304 Free article.

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