Ford HL - Search Results

1

Epithelial-mesenchymal transition in cancer: parallels between normal development and tumor progression.

Micalizzi DS, Farabaugh SM, Ford HL. Micalizzi DS, et al. Among authors: ford hl. J Mammary Gland Biol Neoplasia. 2010 Jun;15(2):117-34. doi: 10.1007/s10911-010-9178-9. Epub 2010 May 19. J Mammary Gland Biol Neoplasia. 2010. PMID: 20490631 Free PMC article. Review.

2

Transcriptional control of the cell cycle in mammary gland development and tumorigenesis.

Coletta RD, Jedlicka P, Gutierrez-Hartmann A, Ford HL. Coletta RD, et al. Among authors: ford hl. J Mammary Gland Biol Neoplasia. 2004 Jan;9(1):39-53. doi: 10.1023/B:JOMG.0000023587.40966.f6. J Mammary Gland Biol Neoplasia. 2004. PMID: 15082917 Review.

3

Six1 overexpression in mammary cells induces genomic instability and is sufficient for malignant transformation.

Coletta RD, Christensen KL, Micalizzi DS, Jedlicka P, Varella-Garcia M, Ford HL. Coletta RD, et al. Among authors: ford hl. Cancer Res. 2008 Apr 1;68(7):2204-13. doi: 10.1158/0008-5472.CAN-07-3141. Cancer Res. 2008. PMID: 18381426

4

Six1 expands the mouse mammary epithelial stem/progenitor cell pool and induces mammary tumors that undergo epithelial-mesenchymal transition.

McCoy EL, Iwanaga R, Jedlicka P, Abbey NS, Chodosh LA, Heichman KA, Welm AL, Ford HL. McCoy EL, et al. Among authors: ford hl. J Clin Invest. 2009 Sep;119(9):2663-77. doi: 10.1172/JCI37691. Epub 2009 Aug 24. J Clin Invest. 2009. PMID: 19726883 Free PMC article.

5

The Six1 homeoprotein induces human mammary carcinoma cells to undergo epithelial-mesenchymal transition and metastasis in mice through increasing TGF-beta signaling.

Micalizzi DS, Christensen KL, Jedlicka P, Coletta RD, Barón AE, Harrell JC, Horwitz KB, Billheimer D, Heichman KA, Welm AL, Schiemann WP, Ford HL. Micalizzi DS, et al. Among authors: ford hl. J Clin Invest. 2009 Sep;119(9):2678-90. doi: 10.1172/JCI37815. Epub 2009 Aug 24. J Clin Invest. 2009. PMID: 19726885 Free PMC article.

6

Epithelial-mesenchymal transition in development and cancer.

Micalizzi DS, Ford HL. Micalizzi DS, et al. Among authors: ford hl. Future Oncol. 2009 Oct;5(8):1129-43. doi: 10.2217/fon.09.94. Future Oncol. 2009. PMID: 19852726 Review.

7

Characterization of the Six1 homeobox gene in normal mammary gland morphogenesis.

Coletta RD, McCoy EL, Burns V, Kawakami K, McManaman JL, Wysolmerski JJ, Ford HL. Coletta RD, et al. Among authors: ford hl. BMC Dev Biol. 2010 Jan 14;10:4. doi: 10.1186/1471-213X-10-4. BMC Dev Biol. 2010. PMID: 20074369 Free PMC article.

8

Mammary gland studies as important contributors to the cause of epithelial mesenchymal plasticity in malignancy.

Ford HL, Thompson EW. Ford HL, et al. J Mammary Gland Biol Neoplasia. 2010 Jun;15(2):113-5. doi: 10.1007/s10911-010-9182-0. J Mammary Gland Biol Neoplasia. 2010. PMID: 20544376 Free article. No abstract available.

9

Homeoprotein Six1 increases TGF-beta type I receptor and converts TGF-beta signaling from suppressive to supportive for tumor growth.

Micalizzi DS, Wang CA, Farabaugh SM, Schiemann WP, Ford HL. Micalizzi DS, et al. Among authors: ford hl. Cancer Res. 2010 Dec 15;70(24):10371-80. doi: 10.1158/0008-5472.CAN-10-1354. Epub 2010 Nov 5. Cancer Res. 2010. PMID: 21056993 Free PMC article.

10

Eya2 is required to mediate the pro-metastatic functions of Six1 via the induction of TGF-β signaling, epithelial-mesenchymal transition, and cancer stem cell properties.

Farabaugh SM, Micalizzi DS, Jedlicka P, Zhao R, Ford HL. Farabaugh SM, et al. Among authors: ford hl. Oncogene. 2012 Feb 2;31(5):552-62. doi: 10.1038/onc.2011.259. Epub 2011 Jun 27. Oncogene. 2012. PMID: 21706047 Free PMC article.

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