Yu SM - Search Results

1

2-Deoxy-D-glucose regulates dedifferentiation through beta-catenin pathway in rabbit articular chondrocytes.

Yu SM, Kim HA, Kim SJ. Yu SM, et al. Exp Mol Med. 2010 Jul 31;42(7):503-13. doi: 10.3858/emm.2010.42.7.051. Exp Mol Med. 2010. PMID: 20530983 Free PMC article.

2

15-deoxy-Delta 12,14-ProstaglandinJ2 regulates dedifferentiation through peroxisome proliferator-activated receptor-gamma-dependent pathway but not COX-2 expression in articular chondrocytes.

Lee JH, Yu SM, Yoon EK, Lee WK, Jung JC, Kim SJ. Lee JH, et al. Among authors: yu sm. J Korean Med Sci. 2007 Oct;22(5):891-7. doi: 10.3346/jkms.2007.22.5.891. J Korean Med Sci. 2007. PMID: 17982241 Free PMC article.

3

Endoplasmic reticulum stress (ER-stress) by 2-deoxy-D-glucose (2DG) reduces cyclooxygenase-2 (COX-2) expression and N-glycosylation and induces a loss of COX-2 activity via a Src kinase-dependent pathway in rabbit articular chondrocytes.

Yu SM, Kim SJ. Yu SM, et al. Exp Mol Med. 2010 Nov 30;42(11):777-86. doi: 10.3858/emm.2010.42.11.079. Exp Mol Med. 2010. PMID: 20926918 Free PMC article.

4

Protein phosphorylation on tyrosine restores expression and glycosylation of cyclooxygenase-2 by 2-deoxy-D-glucose-caused endoplasmic reticulum stress in rabbit articular chondrocyte.

Yu SM, Kim SJ. Yu SM, et al. BMB Rep. 2012 May;45(5):317-22. doi: 10.5483/bmbrep.2012.45.5.317. BMB Rep. 2012. PMID: 22617457 Free article.

5

Sulforaphane inhibits proliferation by causing cell cycle arrest at the G2/M phase in rabbit articular chondrocytes.

Jeong HJ, Yu SM, Jung JC, Kim SJ. Jeong HJ, et al. Among authors: yu sm. Mol Med Rep. 2012 Nov;6(5):1199-203. doi: 10.3892/mmr.2012.1057. Epub 2012 Aug 30. Mol Med Rep. 2012. PMID: 22941232

6

Thymoquinone-induced reactive oxygen species causes apoptosis of chondrocytes via PI3K/Akt and p38kinase pathway.

Yu SM, Kim SJ. Yu SM, et al. Exp Biol Med (Maywood). 2013 Jul;238(7):811-20. doi: 10.1177/1535370213492685. Epub 2013 Jun 20. Exp Biol Med (Maywood). 2013. PMID: 23788172

7

Production of reactive oxygen species by withaferin A causes loss of type collagen expression and COX-2 expression through the PI3K/Akt, p38, and JNK pathways in rabbit articular chondrocytes.

Yu SM, Kim SJ. Yu SM, et al. Exp Cell Res. 2013 Nov 1;319(18):2822-34. doi: 10.1016/j.yexcr.2013.08.026. Epub 2013 Sep 7. Exp Cell Res. 2013. PMID: 24016823

8

Withaferin A-caused production of intracellular reactive oxygen species modulates apoptosis via PI3K/Akt and JNKinase in rabbit articular chondrocytes.

Yu SM, Kim SJ. Yu SM, et al. J Korean Med Sci. 2014 Aug;29(8):1042-53. doi: 10.3346/jkms.2014.29.8.1042. Epub 2014 Jul 30. J Korean Med Sci. 2014. PMID: 25120312 Free PMC article.

9

The thymoquinone-induced production of reactive oxygen species promotes dedifferentiation through the ERK pathway and inflammation through the p38 and PI3K pathways in rabbit articular chondrocytes.

Yu SM, Kim SJ. Yu SM, et al. Int J Mol Med. 2015 Feb;35(2):325-32. doi: 10.3892/ijmm.2014.2014. Epub 2014 Nov 27. Int J Mol Med. 2015. PMID: 25435376 Free PMC article.

10

Salinomycin causes dedifferentiation via the extracellular signal-regulated kinase (ERK) pathway in rabbit articular chondrocytes.

Yu SM, Kim SJ. Yu SM, et al. J Pharmacol Sci. 2015 Feb;127(2):196-202. doi: 10.1016/j.jphs.2014.12.009. Epub 2014 Dec 24. J Pharmacol Sci. 2015. PMID: 25727957 Free article.

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