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Amyloid beta 42 peptide (Abeta42)-lowering compounds directly bind to Abeta and interfere with amyloid precursor protein (APP) transmembrane dimerization.
Richter L, Munter LM, Ness J, Hildebrand PW, Dasari M, Unterreitmeier S, Bulic B, Beyermann M, Gust R, Reif B, Weggen S, Langosch D, Multhaup G. Richter L, et al. Among authors: bulic b. Proc Natl Acad Sci U S A. 2010 Aug 17;107(33):14597-602. doi: 10.1073/pnas.1003026107. Epub 2010 Aug 2. Proc Natl Acad Sci U S A. 2010. PMID: 20679249 Free PMC article.
Presenilin-1 but not amyloid precursor protein mutations present in mouse models of Alzheimer's disease attenuate the response of cultured cells to γ-secretase modulators regardless of their potency and structure.
Hahn S, Brüning T, Ness J, Czirr E, Baches S, Gijsen H, Korth C, Pietrzik CU, Bulic B, Weggen S. Hahn S, et al. Among authors: bulic b. J Neurochem. 2011 Feb;116(3):385-95. doi: 10.1111/j.1471-4159.2010.07118.x. Epub 2010 Dec 13. J Neurochem. 2011. PMID: 21091478 Free article.
Tau protein and tau aggregation inhibitors.
Bulic B, Pickhardt M, Mandelkow EM, Mandelkow E. Bulic B, et al. Neuropharmacology. 2010 Sep-Oct;59(4-5):276-89. doi: 10.1016/j.neuropharm.2010.01.016. Epub 2010 Feb 10. Neuropharmacology. 2010. PMID: 20149808 Review.
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