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Page 1
Cardiac origin of smooth muscle cells in the inflow tract.
Nakano H, Williams E, Hoshijima M, Sasaki M, Minamisawa S, Chien KR, Nakano A. Nakano H, et al. Among authors: minamisawa s. J Mol Cell Cardiol. 2011 Feb;50(2):337-45. doi: 10.1016/j.yjmcc.2010.10.009. Epub 2010 Oct 23. J Mol Cell Cardiol. 2011. PMID: 20974149 Free PMC article.
Heterozygous deletion of sarcolipin maintains normal cardiac function.
Shimura D, Kusakari Y, Sasano T, Nakashima Y, Nakai G, Jiao Q, Jin M, Yokota T, Ishikawa Y, Nakano A, Goda N, Minamisawa S. Shimura D, et al. Among authors: minamisawa s. Am J Physiol Heart Circ Physiol. 2016 Jan 1;310(1):H92-103. doi: 10.1152/ajpheart.00411.2015. Epub 2015 Oct 30. Am J Physiol Heart Circ Physiol. 2016. PMID: 26519028 Free article.
A post-transcriptional compensatory pathway in heterozygous ventricular myosin light chain 2-deficient mice results in lack of gene dosage effect during normal cardiac growth or hypertrophy.
Minamisawa S, Gu Y, Ross J Jr, Chien KR, Chen J. Minamisawa S, et al. J Biol Chem. 1999 Apr 9;274(15):10066-70. doi: 10.1074/jbc.274.15.10066. J Biol Chem. 1999. PMID: 10187786 Free article.
Our previous study of homozygous mutants of the ventricular specific isoform of myosin light chain 2 (mlc-2v) demonstrated that mlc-2v plays an essential role in murine heart development (Chen, J., Kubalak, S. W., Minamisawa, S., Price, R. L., Becker, K. D., …
Our previous study of homozygous mutants of the ventricular specific isoform of myosin light chain 2 (mlc-2v) demonstrated that mlc-2v plays …
113 results