Strøm TB - Search Results

1

The cytoplasmic domain is not involved in directing Class 5 mutant LDL receptors to lysosomal degradation.

Strøm TB, Tveten K, Holla ØL, Cameron J, Berge KE, Leren TP. Strøm TB, et al. Biochem Biophys Res Commun. 2011 May 20;408(4):642-6. doi: 10.1016/j.bbrc.2011.04.077. Epub 2011 Apr 21. Biochem Biophys Res Commun. 2011. PMID: 21531209

2

A chimeric LDL receptor containing the cytoplasmic domain of the transferrin receptor is degraded by PCSK9.

Holla ØL, Strøm TB, Cameron J, Berge KE, Leren TP. Holla ØL, et al. Among authors: strom tb. Mol Genet Metab. 2010 Feb;99(2):149-56. doi: 10.1016/j.ymgme.2009.09.012. Epub 2009 Sep 27. Mol Genet Metab. 2010. PMID: 19828345

3

Loss-of-function mutation R46L in the PCSK9 gene has little impact on the levels of total serum cholesterol in familial hypercholesterolemia heterozygotes.

Strøm TB, Holla ØL, Cameron J, Berge KE, Leren TP. Strøm TB, et al. Clin Chim Acta. 2010 Feb;411(3-4):229-33. doi: 10.1016/j.cca.2009.11.008. Epub 2009 Nov 13. Clin Chim Acta. 2010. PMID: 19917273

4

Disrupted recycling of the low density lipoprotein receptor by PCSK9 is not mediated by residues of the cytoplasmic domain.

Strøm TB, Holla ØL, Tveten K, Cameron J, Berge KE, Leren TP. Strøm TB, et al. Mol Genet Metab. 2010 Sep;101(1):76-80. doi: 10.1016/j.ymgme.2010.05.003. Epub 2010 Jun 9. Mol Genet Metab. 2010. PMID: 20659812

5

Removal of acidic residues of the prodomain of PCSK9 increases its activity towards the LDL receptor.

Holla ØL, Laerdahl JK, Strøm TB, Tveten K, Cameron J, Berge KE, Leren TP. Holla ØL, et al. Among authors: strom tb. Biochem Biophys Res Commun. 2011 Mar 11;406(2):234-8. doi: 10.1016/j.bbrc.2011.02.023. Epub 2011 Feb 13. Biochem Biophys Res Commun. 2011. PMID: 21324305

6

Role of the C-terminal domain of PCSK9 in degradation of the LDL receptors.

Holla ØL, Cameron J, Tveten K, Strøm TB, Berge KE, Laerdahl JK, Leren TP. Holla ØL, et al. Among authors: strom tb. J Lipid Res. 2011 Oct;52(10):1787-94. doi: 10.1194/jlr.M018093. Epub 2011 Jul 19. J Lipid Res. 2011. PMID: 21771976 Free PMC article.

7

Characterization of a naturally occurring degradation product of the LDL receptor.

Tveten K, Strøm TB, Cameron J, Holla ØL, Berge KE, Leren TP. Tveten K, et al. Among authors: strom tb. Mol Genet Metab. 2012 Jan;105(1):149-54. doi: 10.1016/j.ymgme.2011.10.008. Epub 2011 Oct 24. Mol Genet Metab. 2012. PMID: 22078455

8

Characterization of residues in the cytoplasmic domain of the LDL receptor required for exit from the endoplasmic reticulum.

Strøm TB, Tveten K, Holla ØL, Cameron J, Berge KE, Leren TP. Strøm TB, et al. Biochem Biophys Res Commun. 2011 Dec 2;415(4):642-5. doi: 10.1016/j.bbrc.2011.10.127. Epub 2011 Nov 2. Biochem Biophys Res Commun. 2011. PMID: 22079632

9

Interaction between the ligand-binding domain of the LDL receptor and the C-terminal domain of PCSK9 is required for PCSK9 to remain bound to the LDL receptor during endosomal acidification.

Tveten K, Holla ØL, Cameron J, Strøm TB, Berge KE, Laerdahl JK, Leren TP. Tveten K, et al. Among authors: strom tb. Hum Mol Genet. 2012 Mar 15;21(6):1402-9. doi: 10.1093/hmg/ddr578. Epub 2011 Dec 8. Hum Mol Genet. 2012. PMID: 22156580

10

Serum levels of proprotein convertase subtilisin/kexin type 9 in subjects with familial hypercholesterolemia indicate that proprotein convertase subtilisin/kexin type 9 is cleared from plasma by low-density lipoprotein receptor-independent pathways.

Cameron J, Bogsrud MP, Tveten K, Strøm TB, Holven K, Berge KE, Leren TP. Cameron J, et al. Among authors: strom tb. Transl Res. 2012 Aug;160(2):125-30. doi: 10.1016/j.trsl.2012.01.010. Epub 2012 Jan 31. Transl Res. 2012. PMID: 22683370

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