Ulick S - Search Results

1

Defective fasciculata zone function as the mechanism of glucocorticoid-remediable aldosteronism.

Ulick S, Chan CK, Gill JR Jr, Gutkin M, Letcher L, Mantero F, New MI. Ulick S, et al. J Clin Endocrinol Metab. 1990 Nov;71(5):1151-7. doi: 10.1210/jcem-71-5-1151. J Clin Endocrinol Metab. 1990. PMID: 2172271

2

Pathogenesis of the type 2 variant of the syndrome of apparent mineralocorticoid excess.

Ulick S, Tedde R, Mantero F. Ulick S, et al. J Clin Endocrinol Metab. 1990 Jan;70(1):200-6. doi: 10.1210/jcem-70-1-200. J Clin Endocrinol Metab. 1990. PMID: 2403571

3

Measurement of 4 urinary C-18 oxygenated corticosteroids by stable isotope dilution mass fragmentography.

Ulick S, Chan CK, Wang JZ. Ulick S, et al. J Steroid Biochem Mol Biol. 1991 Jan;38(1):59-66. doi: 10.1016/0960-0760(91)90401-p. J Steroid Biochem Mol Biol. 1991. PMID: 1997124

4

Anomalous oxidative cleavage of the side chain of 18-oxocortisol and its tetrahydro metabolite.

Chan CK, Ulick S. Chan CK, et al. Among authors: ulick s. J Steroid Biochem. 1989 Oct;33(4A):605-11. doi: 10.1016/0022-4731(89)90048-4. J Steroid Biochem. 1989. PMID: 2554063

The course of oxidative side chain cleavage of two recently isolated 17 alpha-hydroxy C-18 oxygenated naturally-occurring corticosteroids differed in that 18-hydroxycortisol yielded a 17-ketosteroid (Chu M. D., and Ulick S. (1982) J. biol. Chem. 257, 2218-2224) wher …

The course of oxidative side chain cleavage of two recently isolated 17 alpha-hydroxy C-18 oxygenated naturally-occurring corticosteroids di …

5

A new form of the syndrome of apparent mineralocorticoid excess.

Ulick S, Chan CK, Rao KN, Edassery J, Mantero F. Ulick S, et al. J Steroid Biochem. 1989 Jan;32(1B):209-12. doi: 10.1016/0022-4731(89)90166-0. J Steroid Biochem. 1989. PMID: 2913411

6

Metabolic and blood pressure responses to hydrocortisone in the syndrome of apparent mineralocorticoid excess.

Oberfield SE, Levine LS, Carey RM, Greig F, Ulick S, New MI. Oberfield SE, et al. Among authors: ulick s. J Clin Endocrinol Metab. 1983 Feb;56(2):332-9. doi: 10.1210/jcem-56-2-332. J Clin Endocrinol Metab. 1983. PMID: 6296185

7

A syndrome of apparent mineralocorticoid excess associated with defects in the peripheral metabolism of cortisol.

Ulick S, Levine LS, Gunczler P, Zanconato G, Ramirez LC, Rauh W, Rösler A, Bradlow HL, New MI. Ulick S, et al. J Clin Endocrinol Metab. 1979 Nov;49(5):757-64. doi: 10.1210/jcem-49-5-757. J Clin Endocrinol Metab. 1979. PMID: 226561

8

An abnormality in steroid reductive metabolism in a hypertensive syndrome.

Ulick S, Ramirez LC, New MI. Ulick S, et al. J Clin Endocrinol Metab. 1977 Apr;44(4):799-802. doi: 10.1210/jcem-44-4-799. J Clin Endocrinol Metab. 1977. PMID: 849992

9

Evidence for an unidentified steroid in a child with apparent mineralocorticoid hypertension.

New MI, Levine LS, Biglieri EG, Pareira J, Ulick S. New MI, et al. Among authors: ulick s. J Clin Endocrinol Metab. 1977 May;44(5):924-33. doi: 10.1210/jcem-44-5-924. J Clin Endocrinol Metab. 1977. PMID: 870517

10

The unique steroidogenesis of the aldosteronoma in the differential diagnosis of primary aldosteronism.

Ulick S, Blumenfeld JD, Atlas SA, Wang JZ, Vaughan ED Jr. Ulick S, et al. J Clin Endocrinol Metab. 1993 Apr;76(4):873-8. doi: 10.1210/jcem.76.4.8473399. J Clin Endocrinol Metab. 1993. PMID: 8473399

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