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Increasing cardiac contractility after myocardial infarction exacerbates cardiac injury and pump dysfunction.
Zhang H, Chen X, Gao E, MacDonnell SM, Wang W, Kolpakov M, Nakayama H, Zhang X, Jaleel N, Harris DM, Li Y, Tang M, Berretta R, Leri A, Kajstura J, Sabri A, Koch WJ, Molkentin JD, Houser SR. Zhang H, et al. Among authors: zhang x. Circ Res. 2010 Sep 17;107(6):800-9. doi: 10.1161/CIRCRESAHA.110.219220. Epub 2010 Jul 29. Circ Res. 2010. PMID: 20671241 Free PMC article.
Calcium influx through Cav1.2 is a proximal signal for pathological cardiomyocyte hypertrophy.
Chen X, Nakayama H, Zhang X, Ai X, Harris DM, Tang M, Zhang H, Szeto C, Stockbower K, Berretta RM, Eckhart AD, Koch WJ, Molkentin JD, Houser SR. Chen X, et al. Among authors: zhang x, zhang h. J Mol Cell Cardiol. 2011 Mar;50(3):460-70. doi: 10.1016/j.yjmcc.2010.11.012. Epub 2010 Nov 25. J Mol Cell Cardiol. 2011. PMID: 21111744 Free PMC article.
Transient receptor potential channels contribute to pathological structural and functional remodeling after myocardial infarction.
Makarewich CA, Zhang H, Davis J, Correll RN, Trappanese DM, Hoffman NE, Troupes CD, Berretta RM, Kubo H, Madesh M, Chen X, Gao E, Molkentin JD, Houser SR. Makarewich CA, et al. Among authors: zhang h. Circ Res. 2014 Aug 29;115(6):567-580. doi: 10.1161/CIRCRESAHA.115.303831. Epub 2014 Jul 21. Circ Res. 2014. PMID: 25047165 Free PMC article.
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