James FD - Search Results

1

5-Aminoimidazole-4-carboxamide-1-β-D-ribofuranoside (AICAR) effect on glucose production, but not energy metabolism, is independent of hepatic AMPK in vivo.

Hasenour CM, Ridley DE, Hughey CC, James FD, Donahue EP, Shearer J, Viollet B, Foretz M, Wasserman DH. Hasenour CM, et al. Among authors: james fd. J Biol Chem. 2014 Feb 28;289(9):5950-9. doi: 10.1074/jbc.M113.528232. Epub 2014 Jan 8. J Biol Chem. 2014. PMID: 24403081 Free PMC article.

2

5-Aminoimidazole-4-carboxamide-1-beta-D-ribofuranoside causes acute hepatic insulin resistance in vivo.

Pencek RR, Shearer J, Camacho RC, James FD, Lacy DB, Fueger PT, Donahue EP, Snead W, Wasserman DH. Pencek RR, et al. Among authors: james fd. Diabetes. 2005 Feb;54(2):355-60. doi: 10.2337/diabetes.54.2.355. Diabetes. 2005. PMID: 15677492

3

Portal venous 5-aminoimidazole-4-carboxamide-1-beta-D-ribofuranoside infusion overcomes hyperinsulinemic suppression of endogenous glucose output.

Camacho RC, Pencek RR, Lacy DB, James FD, Donahue EP, Wasserman DH. Camacho RC, et al. Among authors: james fd. Diabetes. 2005 Feb;54(2):373-82. doi: 10.2337/diabetes.54.2.373. Diabetes. 2005. PMID: 15677495

4

5-Aminoimidazole-4-carboxamide-1-beta-D-ribofuranoside renders glucose output by the liver of the dog insensitive to a pharmacological increment in insulin.

Camacho RC, Lacy DB, James FD, Donahue EP, Wasserman DH. Camacho RC, et al. Among authors: james fd. Am J Physiol Endocrinol Metab. 2005 Dec;289(6):E1039-43. doi: 10.1152/ajpendo.00247.2005. Epub 2005 Jul 26. Am J Physiol Endocrinol Metab. 2005. PMID: 16046457 Free article.

5

Integrin α1-null mice exhibit improved fatty liver when fed a high fat diet despite severe hepatic insulin resistance.

Williams AS, Kang L, Zheng J, Grueter C, Bracy DP, James FD, Pozzi A, Wasserman DH. Williams AS, et al. Among authors: james fd. J Biol Chem. 2015 Mar 6;290(10):6546-57. doi: 10.1074/jbc.M114.615716. Epub 2015 Jan 15. J Biol Chem. 2015. PMID: 25593319 Free PMC article.

6

Liver AMP-Activated Protein Kinase Is Unnecessary for Gluconeogenesis but Protects Energy State during Nutrient Deprivation.

Hasenour CM, Ridley DE, James FD, Hughey CC, Donahue EP, Viollet B, Foretz M, Young JD, Wasserman DH. Hasenour CM, et al. Among authors: james fd. PLoS One. 2017 Jan 20;12(1):e0170382. doi: 10.1371/journal.pone.0170382. eCollection 2017. PLoS One. 2017. PMID: 28107516 Free PMC article.

7

Loss of hepatic AMP-activated protein kinase impedes the rate of glycogenolysis but not gluconeogenic fluxes in exercising mice.

Hughey CC, James FD, Bracy DP, Donahue EP, Young JD, Viollet B, Foretz M, Wasserman DH. Hughey CC, et al. Among authors: james fd. J Biol Chem. 2017 Dec 8;292(49):20125-20140. doi: 10.1074/jbc.M117.811547. Epub 2017 Oct 16. J Biol Chem. 2017. PMID: 29038293 Free PMC article.

8

Glycine N-methyltransferase deletion in mice diverts carbon flux from gluconeogenesis to pathways that utilize excess methionine cycle intermediates.

Hughey CC, Trefts E, Bracy DP, James FD, Donahue EP, Wasserman DH. Hughey CC, et al. Among authors: james fd. J Biol Chem. 2018 Jul 27;293(30):11944-11954. doi: 10.1074/jbc.RA118.002568. Epub 2018 Jun 11. J Biol Chem. 2018. PMID: 29891549 Free PMC article.

9

Interaction of insulin and prior exercise in control of hepatic metabolism of a glucose load.

Pencek RR, James F, Lacy DB, Jabbour K, Williams PE, Fueger PT, Wasserman DH. Pencek RR, et al. Diabetes. 2003 Aug;52(8):1897-903. doi: 10.2337/diabetes.52.8.1897. Diabetes. 2003. PMID: 12882903

10

Suppression of endogenous glucose production by mild hyperinsulinemia during exercise is determined predominantly by portal venous insulin.

Camacho RC, Pencek RR, Lacy DB, James FD, Wasserman DH. Camacho RC, et al. Among authors: james fd. Diabetes. 2004 Feb;53(2):285-93. doi: 10.2337/diabetes.53.2.285. Diabetes. 2004. PMID: 14747277

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