Lorenz K - Search Results

1

Erratum: Cardiac RKIP induces a beneficial β-adrenoceptor-dependent positive inotropy.

Schmid E, Neef S, Berlin C, Tomasovic A, Kahlert K, Nordbeck P, Deiss K, Denzinger S, Herrmann S, Wettwer E, Weidendorfer M, Becker D, Schäfer F, Wagner N, Ergün S, Schmitt JP, Katus HA, Weidemann F, Ravens U, Maack C, Hein L, Ertl G, Müller OJ, Maier LS, Lohse MJ, Lorenz K. Schmid E, et al. Among authors: lorenz k. Nat Med. 2016 Feb;22(2):217. doi: 10.1038/nm0216-217c. Nat Med. 2016. PMID: 26845407 No abstract available.

2

Protein kinase C switches the Raf kinase inhibitor from Raf-1 to GRK-2.

Lorenz K, Lohse MJ, Quitterer U. Lorenz K, et al. Nature. 2003 Dec 4;426(6966):574-9. doi: 10.1038/nature02158. Nature. 2003. PMID: 14654844

3

The transcriptional repressor Nab1 is a specific regulator of pathological cardiac hypertrophy.

Buitrago M, Lorenz K, Maass AH, Oberdorf-Maass S, Keller U, Schmitteckert EM, Ivashchenko Y, Lohse MJ, Engelhardt S. Buitrago M, et al. Among authors: lorenz k. Nat Med. 2005 Aug;11(8):837-44. doi: 10.1038/nm1272. Epub 2005 Jul 17. Nat Med. 2005. PMID: 16025126

4

Conformational cross-talk between alpha2A-adrenergic and mu-opioid receptors controls cell signaling.

Vilardaga JP, Nikolaev VO, Lorenz K, Ferrandon S, Zhuang Z, Lohse MJ. Vilardaga JP, et al. Among authors: lorenz k. Nat Chem Biol. 2008 Feb;4(2):126-31. doi: 10.1038/nchembio.64. Epub 2008 Jan 13. Nat Chem Biol. 2008. PMID: 18193048

5

A new type of ERK1/2 autophosphorylation causes cardiac hypertrophy.

Lorenz K, Schmitt JP, Schmitteckert EM, Lohse MJ. Lorenz K, et al. Nat Med. 2009 Jan;15(1):75-83. doi: 10.1038/nm.1893. Epub 2008 Dec 7. Nat Med. 2009. PMID: 19060905

6

Alterations of phospholamban function can exhibit cardiotoxic effects independent of excessive sarcoplasmic reticulum Ca2+-ATPase inhibition.

Schmitt JP, Ahmad F, Lorenz K, Hein L, Schulz S, Asahi M, Maclennan DH, Seidman CE, Seidman JG, Lohse MJ. Schmitt JP, et al. Among authors: lorenz k. Circulation. 2009 Jan 27;119(3):436-44. doi: 10.1161/CIRCULATIONAHA.108.783506. Epub 2009 Jan 12. Circulation. 2009. PMID: 19139388

7

Cardiac hypertrophy: targeting Raf/MEK/ERK1/2-signaling.

Lorenz K, Schmitt JP, Vidal M, Lohse MJ. Lorenz K, et al. Int J Biochem Cell Biol. 2009 Dec;41(12):2351-5. doi: 10.1016/j.biocel.2009.08.002. Epub 2009 Aug 8. Int J Biochem Cell Biol. 2009. PMID: 19666137 Review.

8

Raf kinase inhibitor protein (RKIP) dimer formation controls its target switch from Raf1 to G protein-coupled receptor kinase (GRK) 2.

Deiss K, Kisker C, Lohse MJ, Lorenz K. Deiss K, et al. Among authors: lorenz k. J Biol Chem. 2012 Jul 6;287(28):23407-17. doi: 10.1074/jbc.M112.363812. Epub 2012 May 17. J Biol Chem. 2012. PMID: 22610096 Free PMC article.

9

β-Adrenergic receptor stimulation causes cardiac hypertrophy via a Gβγ/Erk-dependent pathway.

Vidal M, Wieland T, Lohse MJ, Lorenz K. Vidal M, et al. Among authors: lorenz k. Cardiovasc Res. 2012 Nov 1;96(2):255-64. doi: 10.1093/cvr/cvs249. Epub 2012 Jul 27. Cardiovasc Res. 2012. PMID: 22843704

10

Interference with ERK(Thr188) phosphorylation impairs pathological but not physiological cardiac hypertrophy.

Ruppert C, Deiss K, Herrmann S, Vidal M, Oezkur M, Gorski A, Weidemann F, Lohse MJ, Lorenz K. Ruppert C, et al. Among authors: lorenz k. Proc Natl Acad Sci U S A. 2013 Apr 30;110(18):7440-5. doi: 10.1073/pnas.1221999110. Epub 2013 Apr 15. Proc Natl Acad Sci U S A. 2013. PMID: 23589880 Free PMC article.

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