Evered CL - Search Results

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Less is more: low expression of MT1-MMP is optimal to promote migration and tumourigenesis of breast cancer cells.

Cepeda MA, Pelling JJ, Evered CL, Williams KC, Freedman Z, Stan I, Willson JA, Leong HS, Damjanovski S. Cepeda MA, et al. Among authors: evered cl. Mol Cancer. 2016 Oct 18;15(1):65. doi: 10.1186/s12943-016-0547-x. Mol Cancer. 2016. PMID: 27756325 Free PMC article.

The cytoplasmic domain of MT1-MMP is dispensable for migration augmentation but necessary to mediate viability of MCF-7 breast cancer cells.

Cepeda MA, Pelling JJ, Evered CL, Leong HS, Damjanovski S. Cepeda MA, et al. Among authors: evered cl. Exp Cell Res. 2017 Jan 1;350(1):169-183. doi: 10.1016/j.yexcr.2016.11.019. Epub 2016 Nov 23. Exp Cell Res. 2017. PMID: 27889376

Inhibition of MT1-MMP proteolytic function and ERK1/2 signalling influences cell migration and invasion through changes in MMP-2 and MMP-9 levels.

Cepeda MA, Evered CL, Pelling JJH, Damjanovski S. Cepeda MA, et al. Among authors: evered cl. J Cell Commun Signal. 2017 Jun;11(2):167-179. doi: 10.1007/s12079-016-0373-3. Epub 2017 Jan 9. J Cell Commun Signal. 2017. PMID: 28070797 Free PMC article.

Stable expression of α1-antitrypsin Portland in MDA-MB-231 cells increased MT1-MMP and MMP-9 levels, but reduced tumour progression.

Willson JA, Muir CA, Evered CL, Cepeda MA, Damjanovski S. Willson JA, et al. Among authors: evered cl. J Cell Commun Signal. 2018 Jun;12(2):479-488. doi: 10.1007/s12079-017-0407-5. Epub 2017 Aug 29. J Cell Commun Signal. 2018. PMID: 28849349 Free PMC article.

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