Sakakibara Y - Search Results

1

Integrated biology approach reveals molecular and pathological interactions among Alzheimer's Aβ42, Tau, TREM2, and TYROBP in Drosophila models.

Sekiya M, Wang M, Fujisaki N, Sakakibara Y, Quan X, Ehrlich ME, De Jager PL, Bennett DA, Schadt EE, Gandy S, Ando K, Zhang B, Iijima KM. Sekiya M, et al. Among authors: sakakibara y. Genome Med. 2018 Mar 29;10(1):26. doi: 10.1186/s13073-018-0530-9. Genome Med. 2018. PMID: 29598827 Free PMC article.

2

EDEM Function in ERAD Protects against Chronic ER Proteinopathy and Age-Related Physiological Decline in Drosophila.

Sekiya M, Maruko-Otake A, Hearn S, Sakakibara Y, Fujisaki N, Suzuki E, Ando K, Iijima KM. Sekiya M, et al. Among authors: sakakibara y. Dev Cell. 2017 Jun 19;41(6):652-664.e5. doi: 10.1016/j.devcel.2017.05.019. Dev Cell. 2017. PMID: 28633019 Free PMC article.

3

Knockdown of wfs1, a fly homolog of Wolfram syndrome 1, in the nervous system increases susceptibility to age- and stress-induced neuronal dysfunction and degeneration in Drosophila.

Sakakibara Y, Sekiya M, Fujisaki N, Quan X, Iijima KM. Sakakibara Y, et al. PLoS Genet. 2018 Jan 22;14(1):e1007196. doi: 10.1371/journal.pgen.1007196. eCollection 2018 Jan. PLoS Genet. 2018. PMID: 29357349 Free PMC article.

4

Cognitive and emotional alterations in App knock-in mouse models of Aβ amyloidosis.

Sakakibara Y, Sekiya M, Saito T, Saido TC, Iijima KM. Sakakibara Y, et al. BMC Neurosci. 2018 Jul 28;19(1):46. doi: 10.1186/s12868-018-0446-8. BMC Neurosci. 2018. PMID: 30055565 Free PMC article.

5

Amyloid-β plaque formation and reactive gliosis are required for induction of cognitive deficits in App knock-in mouse models of Alzheimer's disease.

Sakakibara Y, Sekiya M, Saito T, Saido TC, Iijima KM. Sakakibara Y, et al. BMC Neurosci. 2019 Mar 20;20(1):13. doi: 10.1186/s12868-019-0496-6. BMC Neurosci. 2019. PMID: 30894120 Free PMC article.

6

Widespread Reduced Density of Noradrenergic Locus Coeruleus Axons in the App Knock-In Mouse Model of Amyloid-β Amyloidosis.

Sakakibara Y, Hirota Y, Ibaraki K, Takei K, Chikamatsu S, Tsubokawa Y, Saito T, Saido TC, Sekiya M, Iijima KM. Sakakibara Y, et al. J Alzheimers Dis. 2021;82(4):1513-1530. doi: 10.3233/JAD-210385. J Alzheimers Dis. 2021. PMID: 34180416 Free PMC article.

7

Distinct brain pathologies associated with Alzheimer's disease biomarker-related phospho-tau 181 and phospho-tau 217 in App knock-in mouse models of amyloid-β amyloidosis.

Hirota Y, Sakakibara Y, Ibaraki K, Takei K, Iijima KM, Sekiya M. Hirota Y, et al. Among authors: sakakibara y. Brain Commun. 2022 Nov 6;4(6):fcac286. doi: 10.1093/braincomms/fcac286. eCollection 2022. Brain Commun. 2022. PMID: 36440096 Free PMC article.

8

Drosophila Toll-9 is induced by aging and neurodegeneration to modulate stress signaling and its deficiency exacerbates tau-mediated neurodegeneration.

Sakakibara Y, Yamashiro R, Chikamatsu S, Hirota Y, Tsubokawa Y, Nishijima R, Takei K, Sekiya M, Iijima KM. Sakakibara Y, et al. iScience. 2023 Jan 13;26(2):105968. doi: 10.1016/j.isci.2023.105968. eCollection 2023 Feb 17. iScience. 2023. PMID: 36718365 Free PMC article.

9

Alzheimer's Disease-Related Phospho-Tau181 Signals Are Localized to Demyelinated Axons of Parvalbumin-Positive GABAergic Interneurons in an App Knock-In Mouse Model of Amyloid-β Pathology.

Hirota Y, Sakakibara Y, Takei K, Nishijima R, Sekiya M, Iijima KM. Hirota Y, et al. Among authors: sakakibara y. J Alzheimers Dis. 2023;93(3):1065-1081. doi: 10.3233/JAD-230121. J Alzheimers Dis. 2023. PMID: 37212118 Free PMC article.

10

Decreased plasma nicotinamide and altered NAD⁺ metabolism in glial cells surrounding Aβ plaques in a mouse model of Alzheimer's disease.

Sekiya M, Sakakibara Y, Hirota Y, Ito N, Chikamatsu S, Takei K, Nishijima R, Iijima KM. Sekiya M, et al. Among authors: sakakibara y. Neurobiol Dis. 2024 Nov;202:106694. doi: 10.1016/j.nbd.2024.106694. Epub 2024 Oct 5. Neurobiol Dis. 2024. PMID: 39374707 Free article.

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