Gallacher D - Search Results

1

Evaluation of cardiac arrhythmic risks using a rabbit model of left ventricular systolic dysfunction.

Hemmeryckx B, Feng Y, Frederix L, Lox M, Trenson S, Vreeken R, Lu HR, Gallacher D, Ni Y, Lijnen HR. Hemmeryckx B, et al. Among authors: gallacher d. Eur J Pharmacol. 2018 Aug 5;832:145-155. doi: 10.1016/j.ejphar.2018.05.026. Epub 2018 May 18. Eur J Pharmacol. 2018. PMID: 29782862

2

Both beta-adrenergic receptor stimulation and cardiac tissue type have important roles in elucidating the functional effects of I(Ks) channel blockers in vitro.

Lu HR, Vlaminckx E, Van De Water A, Gallacher DJ. Lu HR, et al. Among authors: gallacher dj. J Pharmacol Toxicol Methods. 2005 Mar-Apr;51(2):81-90. doi: 10.1016/j.vascn.2004.10.004. J Pharmacol Toxicol Methods. 2005. PMID: 15767201

3

A new method to calculate the beat-to-beat instability of QT duration in drug-induced long QT in anesthetized dogs.

van der Linde H, Van de Water A, Loots W, Van Deuren B, Lu HR, Van Ammel K, Peeters M, Gallacher DJ. van der Linde H, et al. Among authors: gallacher dj. J Pharmacol Toxicol Methods. 2005 Jul-Aug;52(1):168-77. doi: 10.1016/j.vascn.2005.03.005. J Pharmacol Toxicol Methods. 2005. PMID: 15935709

4

Choice of cardiac tissue plays an important role in the evaluation of drug-induced prolongation of the QT interval in vitro in rabbit.

Lu HR, Vlaminckx E, Teisman A, Gallacher DJ. Lu HR, et al. J Pharmacol Toxicol Methods. 2005 Jul-Aug;52(1):90-105. doi: 10.1016/j.vascn.2005.04.007. J Pharmacol Toxicol Methods. 2005. PMID: 15978848

5

Calmodulin antagonist W-7 prevents sparfloxacin-induced early afterdepolarizations (EADs) in isolated rabbit purkinje fibers: importance of beat-to-beat instability of the repolarization.

Lu HR, Vlaminckx E, Van de Water A, Gallacher DJ. Lu HR, et al. J Cardiovasc Electrophysiol. 2006 Apr;17(4):415-22. doi: 10.1111/j.1540-8167.2006.00420.x. J Cardiovasc Electrophysiol. 2006. PMID: 16643366

6

In-vitro experimental models for the risk assessment of antibiotic-induced QT prolongation.

Lu HR, Vlaminckx E, Van de Water A, Rohrbacher J, Hermans A, Gallacher DJ. Lu HR, et al. Eur J Pharmacol. 2006 Dec 28;553(1-3):229-39. doi: 10.1016/j.ejphar.2006.09.035. Epub 2006 Sep 23. Eur J Pharmacol. 2006. Corrected and republished in: Eur J Pharmacol. 2007 Dec 22;577(1-3):222-32. doi: 10.1016/j.ejphar.2007.07.070 PMID: 17054943 Corrected and republished.

7

In vivo mechanisms precipitating torsades de pointes in a canine model of drug-induced long-QT1 syndrome.

Gallacher DJ, Van de Water A, van der Linde H, Hermans AN, Lu HR, Towart R, Volders PG. Gallacher DJ, et al. Cardiovasc Res. 2007 Nov 1;76(2):247-56. doi: 10.1016/j.cardiores.2007.06.019. Epub 2007 Jun 29. Cardiovasc Res. 2007. PMID: 17669388

8

Choice of cardiac tissue in vitro plays an important role in assessing the risk of drug-induced cardiac arrhythmias in human: beyond QT prolongation.

Lu HR, Vlaminckx E, Gallacher DJ. Lu HR, et al. J Pharmacol Toxicol Methods. 2008 Jan-Feb;57(1):1-8. doi: 10.1016/j.vascn.2007.06.005. Epub 2007 Jul 17. J Pharmacol Toxicol Methods. 2008. PMID: 17964190

9

In-vitro experimental models for the risk assessment of antibiotic-induced QT prolongation.

Lu HR, Vlaminckx E, Van de Water A, Rohrbacher J, Hermans A, Gallacher DJ. Lu HR, et al. Eur J Pharmacol. 2007 Dec 22;577(1-3):222-32. doi: 10.1016/j.ejphar.2007.07.070. Eur J Pharmacol. 2007. PMID: 18074444

10

Predicting drug-induced changes in QT interval and arrhythmias: QT-shortening drugs point to gaps in the ICHS7B Guidelines.

Lu HR, Vlaminckx E, Hermans AN, Rohrbacher J, Van Ammel K, Towart R, Pugsley M, Gallacher DJ. Lu HR, et al. Among authors: gallacher dj. Br J Pharmacol. 2008 Aug;154(7):1427-38. doi: 10.1038/bjp.2008.191. Epub 2008 May 19. Br J Pharmacol. 2008. PMID: 18493243 Free PMC article.

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