Hotka M - Search Results

1

The paroxysmal depolarization shift in epilepsy research.

Hotka M, Kubista H. Hotka M, et al. Int J Biochem Cell Biol. 2019 Feb;107:77-81. doi: 10.1016/j.biocel.2018.12.006. Epub 2018 Dec 14. Int J Biochem Cell Biol. 2019. PMID: 30557621 Free PMC article. Review.

2

On the Origin of Paroxysmal Depolarization Shifts: The Contribution of Ca_v1.x Channels as the Common Denominator of a Polymorphous Neuronal Discharge Pattern.

Meyer C, Kettner A, Hochenegg U, Rubi L, Hilber K, Koenig X, Boehm S, Hotka M, Kubista H. Meyer C, et al. Among authors: hotka m. Neuroscience. 2021 Aug 1;468:265-281. doi: 10.1016/j.neuroscience.2021.05.011. Epub 2021 May 18. Neuroscience. 2021. PMID: 34015369 Free article.

3

Ca_v 1.3 channels play a crucial role in the formation of paroxysmal depolarization shifts in cultured hippocampal neurons.

Stiglbauer V, Hotka M, Ruiß M, Hilber K, Boehm S, Kubista H. Stiglbauer V, et al. Among authors: hotka m. Epilepsia. 2017 May;58(5):858-871. doi: 10.1111/epi.13719. Epub 2017 Mar 11. Epilepsia. 2017. PMID: 28295232 Free PMC article.

4

The Paroxysmal Depolarization Shift: Reconsidering Its Role in Epilepsy, Epileptogenesis and Beyond.

Kubista H, Boehm S, Hotka M. Kubista H, et al. Among authors: hotka m. Int J Mol Sci. 2019 Jan 29;20(3):577. doi: 10.3390/ijms20030577. Int J Mol Sci. 2019. PMID: 30699993 Free PMC article. Review.

5

Rescue by 4-phenylbutyrate of several misfolded creatine transporter-1 variants linked to the creatine transporter deficiency syndrome.

El-Kasaby A, Kasture A, Koban F, Hotka M, Asjad HMM, Kubista H, Freissmuth M, Sucic S. El-Kasaby A, et al. Among authors: hotka m. Neuropharmacology. 2019 Dec 15;161:107572. doi: 10.1016/j.neuropharm.2019.03.015. Epub 2019 Mar 15. Neuropharmacology. 2019. PMID: 30885608 Free article.

6

L-type Ca²⁺ channel-mediated Ca²⁺ influx adjusts neuronal mitochondrial function to physiological and pathophysiological conditions.

Hotka M, Cagalinec M, Hilber K, Hool L, Boehm S, Kubista H. Hotka M, et al. Sci Signal. 2020 Feb 11;13(618):eaaw6923. doi: 10.1126/scisignal.aaw6923. Sci Signal. 2020. PMID: 32047116 Free PMC article.

7

Glycerol-3-Phosphate Shuttle Is a Backup System Securing Metabolic Flexibility in Neurons.

Dhoundiyal A, Goeschl V, Boehm S, Kubista H, Hotka M. Dhoundiyal A, et al. Among authors: hotka m. J Neurosci. 2022 Sep 28;42(39):7339-7354. doi: 10.1523/JNEUROSCI.0193-22.2022. J Neurosci. 2022. PMID: 35999055 Free PMC article.

8

Severe hydroxymethylbilane synthase deficiency causes depression-like behavior and mitochondrial dysfunction in a mouse model of homozygous dominant acute intermittent porphyria.

Berger S, Stattmann M, Cicvaric A, Monje FJ, Coiro P, Hotka M, Ricken G, Hainfellner J, Greber-Platzer S, Yasuda M, Desnick RJ, Pollak DD. Berger S, et al. Among authors: hotka m. Acta Neuropathol Commun. 2020 Mar 20;8(1):38. doi: 10.1186/s40478-020-00910-z. Acta Neuropathol Commun. 2020. PMID: 32197664 Free PMC article.

9

Detection of Ligand-binding to Membrane Proteins by Capacitance Measurements.

Burtscher V, Hotka M, Sandtner W. Burtscher V, et al. Among authors: hotka m. Bio Protoc. 2019 Jan 5;9(1):e3138. doi: 10.21769/BioProtoc.3138. Bio Protoc. 2019. PMID: 30680295 Free PMC article.

10

An Electrophysiological Approach to Measure Changes in the Membrane Surface Potential in Real Time.

Burtscher V, Hotka M, Freissmuth M, Sandtner W. Burtscher V, et al. Among authors: hotka m. Biophys J. 2020 Feb 25;118(4):813-825. doi: 10.1016/j.bpj.2019.06.033. Epub 2019 Jul 5. Biophys J. 2020. PMID: 31409481 Free PMC article.

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