MacKinnon RN - Search Results

1

Hemopoietic Cell Kinase amplification with Protein Tyrosine Phosphatase Receptor T depletion leads to polycythemia, aberrant marrow erythoid maturation, and splenomegaly.

Ku M, MacKinnon RN, Wall M, Narayan N, Walkley C, Cheng HC, Campbell LJ, Purton LE, Nandurkar H. Ku M, et al. Among authors: mackinnon rn. Sci Rep. 2019 May 7;9(1):7050. doi: 10.1038/s41598-019-43373-6. Sci Rep. 2019. PMID: 31065022 Free PMC article.

2

Dicentric chromosomes and 20q11.2 amplification in MDS/AML with apparent monosomy 20.

Mackinnon RN, Campbell LJ. Mackinnon RN, et al. Cytogenet Genome Res. 2007;119(3-4):211-20. doi: 10.1159/000112063. Epub 2008 Feb 1. Cytogenet Genome Res. 2007. PMID: 18253031

3

A comparison of two contrasting recurrent isochromosomes 20 found in myelodysplastic syndromes suggests that retention of proximal 20q is a significant factor in myeloid malignancies.

MacKinnon RN, Campbell LJ. MacKinnon RN, et al. Cancer Genet Cytogenet. 2005 Dec;163(2):176-9. doi: 10.1016/j.cancergencyto.2005.06.001. Cancer Genet Cytogenet. 2005. PMID: 16337864

4

The paradox of 20q11.21 amplification in a subset of cases of myeloid malignancy with chromosome 20 deletion.

Mackinnon RN, Selan C, Wall M, Baker E, Nandurkar H, Campbell LJ. Mackinnon RN, et al. Genes Chromosomes Cancer. 2010 Nov;49(11):998-1013. doi: 10.1002/gcc.20806. Genes Chromosomes Cancer. 2010. PMID: 20645416

5

Unbalanced translocations of 20q in AML and MDS often involve interstitial rather than terminal deletions of 20q.

MacKinnon RN, Duivenvoorden HM, Campbell LJ. MacKinnon RN, et al. Cancer Genet. 2011 Mar;204(3):153-61. doi: 10.1016/j.cancergen.2010.12.001. Cancer Genet. 2011. PMID: 21504715

6

A cryptic deletion in 5q31.2 provides further evidence for a minimally deleted region in myelodysplastic syndromes.

MacKinnon RN, Kannourakis G, Wall M, Campbell LJ. MacKinnon RN, et al. Cancer Genet. 2011 Apr;204(4):187-94. doi: 10.1016/j.cancergen.2011.02.001. Cancer Genet. 2011. PMID: 21536236

7

Src family kinases and their role in hematological malignancies.

Ku M, Wall M, MacKinnon RN, Walkley CR, Purton LE, Tam C, Izon D, Campbell L, Cheng HC, Nandurkar H. Ku M, et al. Among authors: mackinnon rn. Leuk Lymphoma. 2015 Mar;56(3):577-86. doi: 10.3109/10428194.2014.907897. Epub 2015 Jan 21. Leuk Lymphoma. 2015. PMID: 24898666 Review.

8

The Dicentric Chromosome dic(20;22) Is a Recurrent Abnormality in Myelodysplastic Syndromes and Is a Product of Telomere Fusion.

MacKinnon RN, Duivenvoorden HM, Campbell LJ, Wall M. MacKinnon RN, et al. Cytogenet Genome Res. 2016;150(3-4):262-272. doi: 10.1159/000456677. Epub 2017 Mar 4. Cytogenet Genome Res. 2016. PMID: 28259884

9

A FISH comparison of variant derivatives of the recurrent dic(17;20) of myelodysplastic syndromes and acute myeloid leukemia: Obligatory retention of genes on 17p and 20q may explain the formation of dicentric chromosomes.

MacKinnon RN, Patsouris C, Chudoba I, Campbell LJ. MacKinnon RN, et al. Genes Chromosomes Cancer. 2007 Jan;46(1):27-36. doi: 10.1002/gcc.20385. Genes Chromosomes Cancer. 2007. PMID: 17048234

10

CGH and SNP array using DNA extracted from fixed cytogenetic preparations and long-term refrigerated bone marrow specimens.

Mackinnon RN, Selan C, Zordan A, Wall M, Nandurkar H, Campbell LJ. Mackinnon RN, et al. Mol Cytogenet. 2012 Feb 2;5:10. doi: 10.1186/1755-8166-5-10. Mol Cytogenet. 2012. PMID: 22300816 Free PMC article.

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