Sekijima Y - Search Results

1

Development of diagnostic antibodies against immunoglobulin heavy chain variable region for heavy chain amyloidosis (AH amyloidosis).

Nakagawa M, Yazaki M, Kametani F, Katoh N, Yoshinaga T, Higuchi K, Sekijima Y. Nakagawa M, et al. Among authors: sekijima y. Pathol Int. 2021 Apr;71(4):245-254. doi: 10.1111/pin.13081. Epub 2021 Mar 13. Pathol Int. 2021. PMID: 33713540

2

No association between apolipoprotein E epsilon4 allele and the age of onset in type I familial amyloid polyneuropathy.

Satoh S, Tokuda T, Ikeda S, Sekijima Y, Yanagisawa N, Hidaka H, Kametani F. Satoh S, et al. Among authors: sekijima y. Neurosci Lett. 1996 Feb 9;204(3):209-11. doi: 10.1016/0304-3940(96)12344-2. Neurosci Lett. 1996. PMID: 8938267

3

Presenilin-1 exists in the axoplasm fraction in the brains of aged Down's syndrome subjects and non-demented individuals.

Sekijima Y, Kametani F, Tanaka K, Okochi M, Usami M, Mori H, Tokuda T, Ikeda S. Sekijima Y, et al. Neurosci Lett. 1999 May 28;267(2):121-4. doi: 10.1016/s0304-3940(99)00348-1. Neurosci Lett. 1999. PMID: 10400227

4

Serum transthyretin monomer in patients with familial amyloid polyneuropathy.

Sekijima Y, Tokuda T, Kametani F, Tanaka K, Maruyama K, Ikeda S. Sekijima Y, et al. Amyloid. 2001 Dec;8(4):257-62. doi: 10.3109/13506120108993822. Amyloid. 2001. PMID: 11791618

5

gamma-Secretase can cleave amyloid precursor protein fragments independent of alpha- and beta-secretase pre-cutting.

Kume H, Sekijima Y, Maruyama K, Kametani F. Kume H, et al. Among authors: sekijima y. Int J Mol Med. 2003 Jul;12(1):57-60. Int J Mol Med. 2003. PMID: 12792809

6

Biochemical characteristics of variant transthyretins causing hereditary leptomeningeal amyloidosis.

Mitsuhashi S, Yazaki M, Tokuda T, Sekijima Y, Washimi Y, Shimizu Y, Ando Y, Benson MD, Ikeda S. Mitsuhashi S, et al. Among authors: sekijima y. Amyloid. 2005 Dec;12(4):216-25. doi: 10.1080/13506120500352404. Amyloid. 2005. PMID: 16399646

7

Amyloidogenic transthyretin Val30Met homozygote showing unusually early-onset familial amyloid polyneuropathy.

Tojo K, Sekijima Y, Machida K, Tsuchiya A, Yazaki M, Ikeda S. Tojo K, et al. Among authors: sekijima y. Muscle Nerve. 2008 Jun;37(6):796-803. doi: 10.1002/mus.21028. Muscle Nerve. 2008. PMID: 18506713

8

Non-senile wild-type transthyretin systemic amyloidosis presenting as bilateral carpal tunnel syndrome.

Kodaira M, Sekijima Y, Tojo K, Tsuchiya A, Yazaki M, Ikeda S, Sekijima Y, Hoshii Y, Tachibana S. Kodaira M, et al. Among authors: sekijima y. J Peripher Nerv Syst. 2008 Jun;13(2):148-50. doi: 10.1111/j.1529-8027.2008.00170.x. J Peripher Nerv Syst. 2008. PMID: 18601659 No abstract available.

9

Wild-type transthyretin significantly contributes to the formation of amyloid fibrils in familial amyloid polyneuropathy patients with amyloidogenic transthyretin Val30Met.

Tsuchiya-Suzuki A, Yazaki M, Kametani F, Sekijima Y, Ikeda S. Tsuchiya-Suzuki A, et al. Among authors: sekijima y. Hum Pathol. 2011 Feb;42(2):236-43. doi: 10.1016/j.humpath.2010.06.014. Epub 2010 Nov 5. Hum Pathol. 2011. PMID: 21056899

10

High prevalence of wild-type transthyretin deposition in patients with idiopathic carpal tunnel syndrome: a common cause of carpal tunnel syndrome in the elderly.

Sekijima Y, Uchiyama S, Tojo K, Sano K, Shimizu Y, Imaeda T, Hoshii Y, Kato H, Ikeda S. Sekijima Y, et al. Hum Pathol. 2011 Nov;42(11):1785-91. doi: 10.1016/j.humpath.2011.03.004. Hum Pathol. 2011. PMID: 21733562 Free article.

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