Liang LJ - Search Results

1

Correction: Hypoxia-induced ZEB1 promotes cervical cancer progression via CCL8-dependent tumour-associated macrophage recruitment.

Chen XJ, Deng YR, Wang ZC, Wei WF, Zhou CF, Zhang YM, Yan RM, Liang LJ, Zhong M, Liang L, Wu S, Wang W. Chen XJ, et al. Among authors: liang lj, liang l. Cell Death Dis. 2022 Mar 17;13(3):247. doi: 10.1038/s41419-022-04706-y. Cell Death Dis. 2022. PMID: 35301295 Free PMC article. No abstract available.

2

Cancer-derived exosomal miR-221-3p promotes angiogenesis by targeting THBS2 in cervical squamous cell carcinoma.

Wu XG, Zhou CF, Zhang YM, Yan RM, Wei WF, Chen XJ, Yi HY, Liang LJ, Fan LS, Liang L, Wu S, Wang W. Wu XG, et al. Among authors: liang lj, liang l. Angiogenesis. 2019 Aug;22(3):397-410. doi: 10.1007/s10456-019-09665-1. Epub 2019 Apr 15. Angiogenesis. 2019. Retraction in: Angiogenesis. 2023 Feb;26(1):201. doi: 10.1007/s10456-022-09864-3 PMID: 30993566 Retracted.

3

Hypoxia-induced ZEB1 promotes cervical cancer progression via CCL8-dependent tumour-associated macrophage recruitment.

Chen XJ, Deng YR, Wang ZC, Wei WF, Zhou CF, Zhang YM, Yan RM, Liang LJ, Zhong M, Liang L, Wu S, Wang W. Chen XJ, et al. Among authors: liang lj, liang l. Cell Death Dis. 2019 Jul 1;10(7):508. doi: 10.1038/s41419-019-1748-1. Cell Death Dis. 2019. PMID: 31263103 Free PMC article.

4

Exosome-derived miR-142-5p remodels lymphatic vessels and induces IDO to promote immune privilege in the tumour microenvironment.

Zhou C, Zhang Y, Yan R, Huang L, Mellor AL, Yang Y, Chen X, Wei W, Wu X, Yu L, Liang L, Zhang D, Wu S, Wang W. Zhou C, et al. Cell Death Differ. 2021 Feb;28(2):715-729. doi: 10.1038/s41418-020-00618-6. Epub 2020 Sep 14. Cell Death Differ. 2021. PMID: 32929219 Free PMC article.

5

Periostin⁺ cancer-associated fibroblasts promote lymph node metastasis by impairing the lymphatic endothelial barriers in cervical squamous cell carcinoma.

Wei WF, Chen XJ, Liang LJ, Yu L, Wu XG, Zhou CF, Wang ZC, Fan LS, Hu Z, Liang L, Wang W. Wei WF, et al. Among authors: liang lj, liang l. Mol Oncol. 2021 Jan;15(1):210-227. doi: 10.1002/1878-0261.12837. Epub 2020 Nov 12. Mol Oncol. 2021. PMID: 33124726 Free PMC article.

6

Cancer-secreted exosomal miR-1468-5p promotes tumor immune escape via the immunosuppressive reprogramming of lymphatic vessels.

Zhou C, Wei W, Ma J, Yang Y, Liang L, Zhang Y, Wang Z, Chen X, Huang L, Wang W, Wu S. Zhou C, et al. Mol Ther. 2021 Apr 7;29(4):1512-1528. doi: 10.1016/j.ymthe.2020.12.034. Epub 2021 Jan 1. Mol Ther. 2021. PMID: 33388421 Free PMC article.

7

A novel lymphatic pattern promotes metastasis of cervical cancer in a hypoxic tumour-associated macrophage-dependent manner.

Chen XJ, Wei WF, Wang ZC, Wang N, Guo CH, Zhou CF, Liang LJ, Wu S, Liang L, Wang W. Chen XJ, et al. Among authors: liang lj, liang l. Angiogenesis. 2021 Aug;24(3):549-565. doi: 10.1007/s10456-020-09766-2. Epub 2021 Jan 23. Angiogenesis. 2021. PMID: 33484377 Free PMC article.

8

Tumor-secreted exosomal Wnt2B activates fibroblasts to promote cervical cancer progression.

Liang LJ, Yang Y, Wei WF, Wu XG, Yan RM, Zhou CF, Chen XJ, Wu S, Wang W, Fan LS. Liang LJ, et al. Oncogenesis. 2021 Mar 17;10(3):30. doi: 10.1038/s41389-021-00319-w. Oncogenesis. 2021. PMID: 33731705 Free PMC article.

9

Cancer-secreted exosomal miR-1468-5p promotes tumor immune escape via the immunosuppressive reprogramming of lymphatic vessels.

Zhou C, Wei W, Ma J, Yang Y, Liang L, Zhang Y, Wang Z, Chen X, Huang L, Wang W, Wu S. Zhou C, et al. Mol Ther. 2022 Feb 2;30(2):976-977. doi: 10.1016/j.ymthe.2021.12.014. Epub 2022 Jan 3. Mol Ther. 2022. PMID: 34982969 Free PMC article. No abstract available.

10

Correction: A novel lymphatic pattern promotes metastasis of cervical cancer in a hypoxic tumour-associated macrophage-dependent manner.

Chen XJ, Wei WF, Wang ZC, Wang N, Guo CH, Zhou CF, Liang LJ, Wu S, Liang L, Wang W. Chen XJ, et al. Among authors: liang lj, liang l. Angiogenesis. 2023 Feb;26(1):187-191. doi: 10.1007/s10456-022-09854-5. Angiogenesis. 2023. PMID: 36087167 Free PMC article. No abstract available.

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