White RE - Search Results

1

Epstein-Barr Virus Genome Deletions in Epstein-Barr Virus-Positive T/NK Cell Lymphoproliferative Diseases.

Wongwiwat W, Fournier B, Bassano I, Bayoumy A, Elgueta Karstegl C, Styles C, Bridges R, Lenoir C, BoutBoul D, Moshous D, Neven B, Kanda T, Morgan RG, White RE, Latour S, Farrell PJ. Wongwiwat W, et al. Among authors: white re. J Virol. 2022 Jun 22;96(12):e0039422. doi: 10.1128/jvi.00394-22. Epub 2022 May 25. J Virol. 2022. PMID: 35612313 Free PMC article.

2

Sequences adjacent to oriP improve the persistence of Epstein-Barr virus-based episomes in B cells.

White RE, Wade-Martins R, James MR. White RE, et al. J Virol. 2001 Nov;75(22):11249-52. doi: 10.1128/JVI.75.22.11249-11252.2001. J Virol. 2001. PMID: 11602767 Free PMC article.

3

Epstein-Barr virus origin of lytic replication mediates association of replicating episomes with promyelocytic leukaemia protein nuclear bodies and replication compartments.

Amon W, White RE, Farrell PJ. Amon W, et al. Among authors: white re. J Gen Virol. 2006 May;87(Pt 5):1133-1137. doi: 10.1099/vir.0.81589-0. J Gen Virol. 2006. PMID: 16603513

4

Mutagenesis of the herpesvirus saimiri terminal repeat region reveals important elements for virus production.

White RE, Carline L, Allday MJ. White RE, et al. J Virol. 2007 Jun;81(12):6765-70. doi: 10.1128/JVI.02579-06. Epub 2007 Apr 11. J Virol. 2007. PMID: 17428860 Free PMC article.

5

Epstein-barr virus latency in B cells leads to epigenetic repression and CpG methylation of the tumour suppressor gene Bim.

Paschos K, Smith P, Anderton E, Middeldorp JM, White RE, Allday MJ. Paschos K, et al. Among authors: white re. PLoS Pathog. 2009 Jun;5(6):e1000492. doi: 10.1371/journal.ppat.1000492. Epub 2009 Jun 26. PLoS Pathog. 2009. PMID: 19557159 Free PMC article.

6

Epigenetic repression of p16(INK4A) by latent Epstein-Barr virus requires the interaction of EBNA3A and EBNA3C with CtBP.

Skalska L, White RE, Franz M, Ruhmann M, Allday MJ. Skalska L, et al. Among authors: white re. PLoS Pathog. 2010 Jun 10;6(6):e1000951. doi: 10.1371/journal.ppat.1000951. PLoS Pathog. 2010. PMID: 20548956 Free PMC article.

7

Extensive co-operation between the Epstein-Barr virus EBNA3 proteins in the manipulation of host gene expression and epigenetic chromatin modification.

White RE, Groves IJ, Turro E, Yee J, Kremmer E, Allday MJ. White RE, et al. PLoS One. 2010 Nov 15;5(11):e13979. doi: 10.1371/journal.pone.0013979. PLoS One. 2010. PMID: 21085583 Free PMC article.

8

Cellular gene expression that correlates with EBER expression in Epstein-Barr Virus-infected lymphoblastoid cell lines.

Gregorovic G, Bosshard R, Karstegl CE, White RE, Pattle S, Chiang AK, Dittrich-Breiholz O, Kracht M, Russ R, Farrell PJ. Gregorovic G, et al. Among authors: white re. J Virol. 2011 Apr;85(7):3535-45. doi: 10.1128/JVI.02086-10. Epub 2011 Jan 19. J Virol. 2011. PMID: 21248031 Free PMC article.

9

Latent Epstein-Barr virus can inhibit apoptosis in B cells by blocking the induction of NOXA expression.

Yee J, White RE, Anderton E, Allday MJ. Yee J, et al. Among authors: white re. PLoS One. 2011;6(12):e28506. doi: 10.1371/journal.pone.0028506. Epub 2011 Dec 9. PLoS One. 2011. PMID: 22174825 Free PMC article.

10

Induction of p16(INK4a) is the major barrier to proliferation when Epstein-Barr virus (EBV) transforms primary B cells into lymphoblastoid cell lines.

Skalska L, White RE, Parker GA, Turro E, Sinclair AJ, Paschos K, Allday MJ. Skalska L, et al. Among authors: white re. PLoS Pathog. 2013 Feb;9(2):e1003187. doi: 10.1371/journal.ppat.1003187. Epub 2013 Feb 21. PLoS Pathog. 2013. PMID: 23436997 Free PMC article.

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