Oesterreich S - Search Results

1

Loss of E-cadherin Induces IGF1R Activation and Reveals a Targetable Pathway in Invasive Lobular Breast Carcinoma.

Elangovan A, Hooda J, Savariau L, Puthanmadhomnarayanan S, Yates ME, Chen J, Brown DD, McAuliffe PF, Oesterreich S, Atkinson JM, Lee AV. Elangovan A, et al. Among authors: oesterreich s. Mol Cancer Res. 2022 Sep 2;20(9):1405-1419. doi: 10.1158/1541-7786.MCR-22-0090. Mol Cancer Res. 2022. PMID: 35665642 Free PMC article.

2

Novel nuclear matrix protein HET binds to and influences activity of the HSP27 promoter in human breast cancer cells.

Oesterreich S, Lee AV, Sullivan TM, Samuel SK, Davie JR, Fuqua SA. Oesterreich S, et al. J Cell Biochem. 1997 Nov 1;67(2):275-86. J Cell Biochem. 1997. PMID: 9328833

3

Insulin-like growth factor I-induced degradation of insulin receptor substrate 1 is mediated by the 26S proteasome and blocked by phosphatidylinositol 3'-kinase inhibition.

Lee AV, Gooch JL, Oesterreich S, Guler RL, Yee D. Lee AV, et al. Among authors: oesterreich s. Mol Cell Biol. 2000 Mar;20(5):1489-96. doi: 10.1128/MCB.20.5.1489-1496.2000. Mol Cell Biol. 2000. PMID: 10669726 Free PMC article.

4

HET/SAF-B overexpression causes growth arrest and multinuclearity and is associated with aneuploidy in human breast cancer.

Townson SM, Sullivan T, Zhang Q, Clark GM, Osborne CK, Lee AV, Oesterreich S. Townson SM, et al. Among authors: oesterreich s. Clin Cancer Res. 2000 Sep;6(9):3788-96. Clin Cancer Res. 2000. PMID: 10999774

5

Inhibition of oestrogen receptor activity by the co-repressor HET/SAF-B is relieved by blockade of histone deacetylase activity.

Oesterreich S, Zhang QP, Lee AV. Oesterreich S, et al. Eur J Cancer. 2000 Sep;36 Suppl 4:S43-4. doi: 10.1016/s0959-8049(00)00221-5. Eur J Cancer. 2000. PMID: 11056314 No abstract available.

6

High rates of loss of heterozygosity on chromosome 19p13 in human breast cancer.

Oesterreich S, Allredl DC, Mohsin SK, Zhang Q, Wong H, Lee AV, Osborne CK, O'Connell P. Oesterreich S, et al. Br J Cancer. 2001 Feb;84(4):493-8. doi: 10.1054/bjoc.2000.1606. Br J Cancer. 2001. PMID: 11207044 Free PMC article.

7

Re-expression of estrogen receptor alpha in estrogen receptor alpha-negative MCF-7 cells restores both estrogen and insulin-like growth factor-mediated signaling and growth.

Oesterreich S, Zhang P, Guler RL, Sun X, Curran EM, Welshons WV, Osborne CK, Lee AV. Oesterreich S, et al. Cancer Res. 2001 Aug 1;61(15):5771-7. Cancer Res. 2001. PMID: 11479214

8

Cross-talk among estrogen receptor, epidermal growth factor, and insulin-like growth factor signaling in breast cancer.

Lee AV, Cui X, Oesterreich S. Lee AV, et al. Among authors: oesterreich s. Clin Cancer Res. 2001 Dec;7(12 Suppl):4429s-4435s; discussion 4411s-4412s. Clin Cancer Res. 2001. PMID: 11916236 Review.

9

New mechanisms of signal transduction inhibitor action: receptor tyrosine kinase down-regulation and blockade of signal transactivation.

Lee AV, Schiff R, Cui X, Sachdev D, Yee D, Gilmore AP, Streuli CH, Oesterreich S, Hadsell DL. Lee AV, et al. Among authors: oesterreich s. Clin Cancer Res. 2003 Jan;9(1 Pt 2):516S-23S. Clin Cancer Res. 2003. PMID: 12538509 Review.

10

Insulin-like growth factor-I inhibits progesterone receptor expression in breast cancer cells via the phosphatidylinositol 3-kinase/Akt/mammalian target of rapamycin pathway: progesterone receptor as a potential indicator of growth factor activity in breast cancer.

Cui X, Zhang P, Deng W, Oesterreich S, Lu Y, Mills GB, Lee AV. Cui X, et al. Among authors: oesterreich s. Mol Endocrinol. 2003 Apr;17(4):575-88. doi: 10.1210/me.2002-0318. Epub 2003 Jan 2. Mol Endocrinol. 2003. PMID: 12554765

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