Giacca A - Search Results

1

β-Cell Stress Pathways in Diabetes: Potential Targets for Therapy?

Rahman SMN, Giacca A. Rahman SMN, et al. Among authors: giacca a. Endocrinology. 2022 Dec 19;164(2):bqac211. doi: 10.1210/endocr/bqac211. Endocrinology. 2022. PMID: 36534981 No abstract available.

2

Susceptibility to fatty acid-induced β-cell dysfunction is enhanced in prediabetic diabetes-prone biobreeding rats: a potential link between β-cell lipotoxicity and islet inflammation.

Tang C, Naassan AE, Chamson-Reig A, Koulajian K, Goh TT, Yoon F, Oprescu AI, Ghanim H, Lewis GF, Dandona P, Donath MY, Ehses JA, Arany E, Giacca A. Tang C, et al. Among authors: giacca a. Endocrinology. 2013 Jan;154(1):89-101. doi: 10.1210/en.2012-1720. Epub 2012 Nov 13. Endocrinology. 2013. PMID: 23150493

3

Combined Hyperglycemia- and Hyperinsulinemia-Induced Insulin Resistance in Adipocytes Is Associated With Dual Signaling Defects Mediated by PKC-ζ.

Lu H, Bogdanovic E, Yu Z, Cho C, Liu L, Ho K, Guo J, Yeung LSN, Lehmann R, Hundal HS, Giacca A, Fantus IG. Lu H, et al. Among authors: giacca a. Endocrinology. 2018 Apr 1;159(4):1658-1677. doi: 10.1210/en.2017-00312. Endocrinology. 2018. PMID: 29370351 Free PMC article.

4

Glucose-Induced β-Cell Dysfunction In Vivo: Evidence for a Causal Role of C-jun N-terminal Kinase Pathway.

Tang C, Yeung LSN, Koulajian K, Zhang L, Tai K, Volchuk A, Giacca A. Tang C, et al. Among authors: giacca a. Endocrinology. 2018 Nov 1;159(11):3643-3654. doi: 10.1210/en.2018-00566. Endocrinology. 2018. PMID: 30215691 Free PMC article.

5

NOD1: An Interface Between Innate Immunity and Insulin Resistance.

Rivers SL, Klip A, Giacca A. Rivers SL, et al. Among authors: giacca a. Endocrinology. 2019 May 1;160(5):1021-1030. doi: 10.1210/en.2018-01061. Endocrinology. 2019. PMID: 30807635 Free PMC article. Review.

6

β-Cell Insulin Resistance Plays a Causal Role in Fat-Induced β-Cell Dysfunction In Vitro and In Vivo.

Ivovic A, Yung JHM, Oprescu AI, Vlavcheski F, Mori Y, Rahman SMN, Ye W, Eversley JA, Wheeler MB, Woo M, Tsiani E, Giacca A. Ivovic A, et al. Among authors: giacca a. Endocrinology. 2024 Mar 29;165(5):bqae044. doi: 10.1210/endocr/bqae044. Endocrinology. 2024. PMID: 38578954 Free PMC article.

7

Insulin-like growth factor-I and insulin have no differential effects on glucose production and utilization under conditions of hyperglycemia.

Giacca A, Fisher SJ, Shi ZQ, Gupta R, Lickley HL, Skottner A, Anderson GH, Efendic S, Vranic M. Giacca A, et al. Endocrinology. 1994 May;134(5):2251-8. doi: 10.1210/endo.134.5.8156929. Endocrinology. 1994. PMID: 8156929

8

Direct and indirect effects of insulin in suppressing glucose production in depancreatized dogs: role of glucagon.

Giacca A, Fisher SJ, McCall RH, Shi ZQ, Vranic M. Giacca A, et al. Endocrinology. 1997 Mar;138(3):999-1007. doi: 10.1210/endo.138.3.5025. Endocrinology. 1997. PMID: 9048601

9

Low-dose IGF-I has no selective advantage over insulin in regulating glucose metabolism in hyperglycemic depancreatized dogs.

Fisher SJ, Shi ZQ, Lickley HL, Efendic S, Vranic M, Giacca A. Fisher SJ, et al. Among authors: giacca a. J Endocrinol. 2001 Jan;168(1):49-58. doi: 10.1677/joe.0.1680049. J Endocrinol. 2001. PMID: 11139769

10

Hyperinsulinemia, but not other factors associated with insulin resistance, acutely enhances colorectal epithelial proliferation in vivo.

Tran TT, Naigamwalla D, Oprescu AI, Lam L, McKeown-Eyssen G, Bruce WR, Giacca A. Tran TT, et al. Among authors: giacca a. Endocrinology. 2006 Apr;147(4):1830-7. doi: 10.1210/en.2005-1012. Epub 2006 Jan 12. Endocrinology. 2006. PMID: 16410309

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