Que L - Search Results

1

ECSIT-X4 is Required for Preventing Pressure Overload-Induced Cardiac Hypertrophy via Regulating Mitochondrial STAT3.

Lu X, Tong T, Sun H, Chen Y, Shao Y, Shi P, Que L, Liu L, Zhu G, Chen Q, Li C, Li J, Yang S, Li Y. Lu X, et al. Among authors: que l. Adv Sci (Weinh). 2025 Jan 2:e2414358. doi: 10.1002/advs.202414358. Online ahead of print. Adv Sci (Weinh). 2025. PMID: 39746855

2

Pellino1-mediated TGF-β1 synthesis contributes to mechanical stress induced cardiac fibroblast activation.

Song J, Zhu Y, Li J, Liu J, Gao Y, Ha T, Que L, Liu L, Zhu G, Chen Q, Xu Y, Li C, Li Y. Song J, et al. Among authors: que l. J Mol Cell Cardiol. 2015 Feb;79:145-56. doi: 10.1016/j.yjmcc.2014.11.006. Epub 2014 Nov 13. J Mol Cell Cardiol. 2015. PMID: 25446187

3

The TIR/BB-loop mimetic AS-1 attenuates mechanical stress-induced cardiac fibroblast activation and paracrine secretion via modulation of large tumor suppressor kinase 1.

Fan M, Song J, He Y, Shen X, Li J, Que L, Zhu G, Zhu Q, Cai X, Ha T, Chen Q, Xu Y, Li C, Li Y. Fan M, et al. Among authors: que l. Biochim Biophys Acta. 2016 Jun;1862(6):1191-202. doi: 10.1016/j.bbadis.2016.03.002. Epub 2016 Mar 7. Biochim Biophys Acta. 2016. PMID: 26964796 Free article.

4

Peli1 induction impairs cardiac microvascular endothelium through Hsp90 dissociation from IRE1α.

Zhao Q, Yang J, Chen H, Li J, Que L, Zhu G, Liu L, Ha T, Chen Q, Li C, Xu Y, Li Y. Zhao Q, et al. Among authors: que l. Biochim Biophys Acta Mol Basis Dis. 2019 Oct 1;1865(10):2606-2617. doi: 10.1016/j.bbadis.2019.06.017. Epub 2019 Jun 29. Biochim Biophys Acta Mol Basis Dis. 2019. PMID: 31260751 Free article.

5

Triad3A attenuates pathological cardiac hypertrophy involving the augmentation of ubiquitination-mediated degradation of TLR4 and TLR9.

Lu X, He Y, Tang C, Wang X, Que L, Zhu G, Liu L, Ha T, Chen Q, Li C, Xu Y, Li J, Li Y. Lu X, et al. Among authors: que l. Basic Res Cardiol. 2020 Feb 1;115(2):19. doi: 10.1007/s00395-020-0779-1. Basic Res Cardiol. 2020. PMID: 32008145

6

Peli1 contributes to myocardial ischemia/reperfusion injury by impairing autophagy flux via its E3 ligase mediated ubiquitination of P62.

Yang J, Tong T, Zhu C, Zhou M, Jiang Y, Chen H, Que L, Liu L, Zhu G, Ha T, Chen Q, Li C, Xu Y, Li J, Li Y. Yang J, et al. Among authors: que l. J Mol Cell Cardiol. 2022 Dec;173:30-46. doi: 10.1016/j.yjmcc.2022.09.004. Epub 2022 Sep 27. J Mol Cell Cardiol. 2022. PMID: 36179399

7

Cardiomyocyte-specific Peli1 contributes to the pressure overload-induced cardiac fibrosis through miR-494-3p-dependent exosomal communication.

Tang C, Hou YX, Shi PX, Zhu CH, Lu X, Wang XL, Que LL, Zhu GQ, Liu L, Chen Q, Li CF, Xu Y, Li JT, Li YH. Tang C, et al. Among authors: que ll. FASEB J. 2023 Jan;37(1):e22699. doi: 10.1096/fj.202200597R. FASEB J. 2023. PMID: 36520055

8

HNF4α ubiquitination mediated by Peli1 impairs FAO and accelerates pressure overload-induced myocardial hypertrophy.

Hou Y, Shi P, Du H, Zhu C, Tang C, Que L, Zhu G, Liu L, Chen Q, Li C, Shao G, Li Y, Li J. Hou Y, et al. Among authors: que l. Cell Death Dis. 2024 Feb 12;15(2):135. doi: 10.1038/s41419-024-06470-7. Cell Death Dis. 2024. PMID: 38346961 Free PMC article.

9

Silencing of Pellino1 improves post-infarct cardiac dysfunction and attenuates left ventricular remodelling in mice.

Wu W, Hu Y, Li J, Zhu W, Ha T, Que L, Liu L, Zhu Q, Chen Q, Xu Y, Li C, Li Y. Wu W, et al. Among authors: que l. Cardiovasc Res. 2014 Apr 1;102(1):46-55. doi: 10.1093/cvr/cvu007. Epub 2014 Jan 16. Cardiovasc Res. 2014. PMID: 24442869

10

Peli1 deletion in macrophages attenuates myocardial ischemia/reperfusion injury by suppressing M1 polarization.

Chen H, Hou Y, Zhai Y, Yang J, Que L, Liu J, Lu L, Ha T, Li C, Xu Y, Li J, Li Y. Chen H, et al. Among authors: que l. J Leukoc Biol. 2023 Feb 1;113(2):95-108. doi: 10.1093/jleuko/qiac012. J Leukoc Biol. 2023. PMID: 36822176

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