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Studies of the differentiation of promyelocytic cells by phorbol ester. II. A methylation inhibitor, 3-deazaadenosine, inhibits the induction of specific differentiation proteins. Lack of effect on early and late phosphorylation events.
Feuerstein N, Cooper HL. Feuerstein N, et al. Among authors: cooper hl. Biochim Biophys Acta. 1984 Apr 5;781(3):247-56. doi: 10.1016/0167-4781(84)90090-3. Biochim Biophys Acta. 1984. PMID: 6584181
Cells of the promyelocytic leukemic line, HL-60, differentiate into macrophage-like cells in response to phorbol-12-myristate-13-acetate (phorbol ester). ...Most significantly, 3-deazaadenosine did not inhibit the induction of phosphorylation in pp17 and pp27, which was pr …
Cells of the promyelocytic leukemic line, HL-60, differentiate into macrophage-like cells in response to phorbol-12-myristate-13-acet …
Identification and characterization of the nonphosphorylated precursor of pp17, a phosphoprotein associated with phorbol ester induction of growth arrest and monocytic differentiation in HL-60 promyelocytic leukemia cells.
Braverman R, Bhattacharya B, Feuerstein N, Cooper HL. Braverman R, et al. Among authors: cooper hl. J Biol Chem. 1986 Oct 25;261(30):14342-8. J Biol Chem. 1986. PMID: 3464595 Free article.
Treatment of HL-60 promyelocytic leukemia cells with the tumor-promoting phorbol ester, 12-O-tetradecanoylphorbol-13-acetate (TPA), causes rapid phosphorylation and dephosphorylation of pp17, a 17-20-kDa, pI 5.5 cytosolic protein, as an early event in a response sequence l …
Treatment of HL-60 promyelocytic leukemia cells with the tumor-promoting phorbol ester, 12-O-tetradecanoylphorbol-13-acetate (TPA), c …
302 results