Sah VP - Search Results

1

A subset of p53-deficient embryos exhibit exencephaly.

Sah VP, Attardi LD, Mulligan GJ, Williams BO, Bronson RT, Jacks T. Sah VP, et al. Nat Genet. 1995 Jun;10(2):175-80. doi: 10.1038/ng0695-175. Nat Genet. 1995. PMID: 7663512

2

Altered cell cycle kinetics, gene expression, and G1 restriction point regulation in Rb-deficient fibroblasts.

Herrera RE, Sah VP, Williams BO, Mäkelä TP, Weinberg RA, Jacks T. Herrera RE, et al. Among authors: sah vp. Mol Cell Biol. 1996 May;16(5):2402-7. doi: 10.1128/MCB.16.5.2402. Mol Cell Biol. 1996. PMID: 8628308 Free PMC article.

3

Rho is required for Galphaq and alpha1-adrenergic receptor signaling in cardiomyocytes. Dissociation of Ras and Rho pathways.

Sah VP, Hoshijima M, Chien KR, Brown JH. Sah VP, et al. J Biol Chem. 1996 Dec 6;271(49):31185-90. doi: 10.1074/jbc.271.49.31185. J Biol Chem. 1996. PMID: 8940118 Free article.

4

The cardiac-specific nuclear delta(B) isoform of Ca2+/calmodulin-dependent protein kinase II induces hypertrophy and dilated cardiomyopathy associated with increased protein phosphatase 2A activity.

Zhang T, Johnson EN, Gu Y, Morissette MR, Sah VP, Gigena MS, Belke DD, Dillmann WH, Rogers TB, Schulman H, Ross J Jr, Brown JH. Zhang T, et al. Among authors: sah vp. J Biol Chem. 2002 Jan 11;277(2):1261-7. doi: 10.1074/jbc.M108525200. Epub 2001 Nov 2. J Biol Chem. 2002. PMID: 11694533 Free article.

5

The MEKK-JNK pathway is stimulated by alpha1-adrenergic receptor and ras activation and is associated with in vitro and in vivo cardiac hypertrophy.

Ramirez MT, Sah VP, Zhao XL, Hunter JJ, Chien KR, Brown JH. Ramirez MT, et al. Among authors: sah vp. J Biol Chem. 1997 May 30;272(22):14057-61. doi: 10.1074/jbc.272.22.14057. J Biol Chem. 1997. PMID: 9162028 Free article.

6

Cardiac muscle cell hypertrophy and apoptosis induced by distinct members of the p38 mitogen-activated protein kinase family.

Wang Y, Huang S, Sah VP, Ross J Jr, Brown JH, Han J, Chien KR. Wang Y, et al. Among authors: sah vp. J Biol Chem. 1998 Jan 23;273(4):2161-8. doi: 10.1074/jbc.273.4.2161. J Biol Chem. 1998. PMID: 9442057 Free article.

7

Cardiac hypertrophy induced by mitogen-activated protein kinase kinase 7, a specific activator for c-Jun NH2-terminal kinase in ventricular muscle cells.

Wang Y, Su B, Sah VP, Brown JH, Han J, Chien KR. Wang Y, et al. Among authors: sah vp. J Biol Chem. 1998 Mar 6;273(10):5423-6. doi: 10.1074/jbc.273.10.5423. J Biol Chem. 1998. PMID: 9488659 Free article.

8

The low molecular weight GTPase Rho regulates myofibril formation and organization in neonatal rat ventricular myocytes. Involvement of Rho kinase.

Hoshijima M, Sah VP, Wang Y, Chien KR, Brown JH. Hoshijima M, et al. Among authors: sah vp. J Biol Chem. 1998 Mar 27;273(13):7725-30. doi: 10.1074/jbc.273.13.7725. J Biol Chem. 1998. PMID: 9516480 Free article.

9

Enhanced Galphaq signaling: a common pathway mediates cardiac hypertrophy and apoptotic heart failure.

Adams JW, Sakata Y, Davis MG, Sah VP, Wang Y, Liggett SB, Chien KR, Brown JH, Dorn GW 2nd. Adams JW, et al. Among authors: sah vp. Proc Natl Acad Sci U S A. 1998 Aug 18;95(17):10140-5. doi: 10.1073/pnas.95.17.10140. Proc Natl Acad Sci U S A. 1998. PMID: 9707614 Free PMC article.

10

The role of Rho in G protein-coupled receptor signal transduction.

Sah VP, Seasholtz TM, Sagi SA, Brown JH. Sah VP, et al. Annu Rev Pharmacol Toxicol. 2000;40:459-89. doi: 10.1146/annurev.pharmtox.40.1.459. Annu Rev Pharmacol Toxicol. 2000. PMID: 10836144 Review.

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