A potent activator of HIV-1 replication is present in the genital tract of a subset of HIV-1-infected and uninfected women

AIDS. 1997 Sep;11(11):1319-26. doi: 10.1097/00002030-199711000-00005.

Abstract

Objective and design: To determine whether the female genital tract contains factors that affect HIV-1 replication. Cervicovaginal lavage (CVL) samples were collected from HIV-1-seropositive and seronegative women and added to cell cultures.

Methods: HIV p24 production was used to measure the effects of CVL on replication of HIVMN in a T-cell line, of a primary isolate in peripheral blood mononuclear cells, or on HIV expression by the latently-infected monocytic U1 cell line. The effects of CVL on the HIV long terminal repeat (LTR) were determined in 1G5 T cells by measuring luciferase activity.

Results: Increased replication of HIVMN and a primary isolate were observed in T cells cultured with CVL samples from three out of 38 HIV-infected women, one out of four uninfected high-risk women, and none of 12 low-risk women. The CVL factor increased replication by enhancing virus expression via activation of the HIV LTR. The HIV-inducing activity was highly stable to heat but was sensitive to proteases, indicating that the activity was distinct from heat-labile cytokines including tumour necrosis factor-alpha.

Conclusions: This is the first study to show that a factor which can stimulate HIV-1 replication is present at biologically active levels in the reproductive tract of women. This factor could potentially affect sexual or vertical transmission of HIV-1 by altering genital tract virus load or virus expression.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Cells, Cultured
  • Endopeptidases / pharmacology
  • Female
  • Genitalia, Female / metabolism*
  • Genitalia, Female / virology*
  • HIV Core Protein p24 / analysis
  • HIV Core Protein p24 / metabolism
  • HIV Infections / metabolism*
  • HIV Infections / transmission
  • HIV Infections / virology
  • HIV Long Terminal Repeat / genetics
  • HIV Seronegativity
  • HIV Seropositivity
  • HIV-1 / growth & development*
  • HIV-1 / pathogenicity
  • Heating
  • Humans
  • Infectious Disease Transmission, Vertical
  • Monocytes / virology
  • T-Lymphocytes / virology
  • Therapeutic Irrigation
  • Transcription, Genetic
  • Tumor Necrosis Factor-alpha / metabolism
  • Tumor Necrosis Factor-alpha / physiology
  • Virus Latency

Substances

  • HIV Core Protein p24
  • Tumor Necrosis Factor-alpha
  • Endopeptidases