R37 HL033921-28/HL/NHLBI NIH HHS/United States[Grants and Funding] - Search Results

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Year	Number of Results
2010	1
2012	2
2025	0

Page 1

Increasing cardiac contractility after myocardial infarction exacerbates cardiac injury and pump dysfunction.

Zhang H, Chen X, Gao E, MacDonnell SM, Wang W, Kolpakov M, Nakayama H, Zhang X, Jaleel N, Harris DM, Li Y, Tang M, Berretta R, Leri A, Kajstura J, Sabri A, Koch WJ, Molkentin JD, Houser SR. Zhang H, et al. Circ Res. 2010 Sep 17;107(6):800-9. doi: 10.1161/CIRCRESAHA.110.219220. Epub 2010 Jul 29. Circ Res. 2010. PMID: 20671241 Free PMC article.

Hyperphosphorylation of the cardiac ryanodine receptor at serine 2808 is not involved in cardiac dysfunction after myocardial infarction.

Zhang H, Makarewich CA, Kubo H, Wang W, Duran JM, Li Y, Berretta RM, Koch WJ, Chen X, Gao E, Valdivia HH, Houser SR. Zhang H, et al. Circ Res. 2012 Mar 16;110(6):831-40. doi: 10.1161/CIRCRESAHA.111.255158. Epub 2012 Feb 2. Circ Res. 2012. PMID: 22302785 Free PMC article.

A caveolae-targeted L-type Ca²+ channel antagonist inhibits hypertrophic signaling without reducing cardiac contractility.

Makarewich CA, Correll RN, Gao H, Zhang H, Yang B, Berretta RM, Rizzo V, Molkentin JD, Houser SR. Makarewich CA, et al. Circ Res. 2012 Mar 2;110(5):669-74. doi: 10.1161/CIRCRESAHA.111.264028. Epub 2012 Feb 2. Circ Res. 2012. PMID: 22302787 Free PMC article.

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